Genetic Susceptibility to Hypertension-Induced Renal Injury.
作者信息
Roman Richard J, Fan Fan
机构信息
From the Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson.
出版信息
Hypertension. 2018 Apr;71(4):559-560. doi: 10.1161/HYPERTENSIONAHA.118.10773. Epub 2018 Feb 5.
Abstract
摘要
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本文引用的文献
1
Susceptibility to Hypertensive Renal Disease in the Spontaneously Hypertensive Rat Is Influenced by 2 Loci Affecting Blood Pressure and Immunoglobulin Repertoire.自发性高血压大鼠发生高血压性肾病的易感性受影响血压和免疫球蛋白库的 2 个基因座的影响。
Hypertension. 2018 Apr;71(4):700-708. doi: 10.1161/HYPERTENSIONAHA.117.10593. Epub 2018 Feb 5.
2
Hypertensive renal injury is associated with gene variation affecting immune signaling.高血压性肾损伤与影响免疫信号传导的基因变异有关。
Circ Cardiovasc Genet. 2014 Dec;7(6):903-10. doi: 10.1161/CIRCGENETICS.114.000533. Epub 2014 Nov 3.
3
Critical blood pressure threshold dependence of hypertensive injury and repair in a malignant nephrosclerosis model.恶性肾硬化模型中高血压损伤与修复对临界血压阈值的依赖性
Hypertension. 2014 Oct;64(4):801-7. doi: 10.1161/HYPERTENSIONAHA.114.03609. Epub 2014 Jun 23.
4
Molecular mechanisms of renal blood flow autoregulation.肾血流自身调节的分子机制。
Curr Vasc Pharmacol. 2014;12(6):845-58. doi: 10.2174/15701611113116660149.
5
Mapping genetic determinants of kidney damage in rat models.绘制大鼠模型肾脏损伤的遗传决定因素图谱。
Hypertens Res. 2012 Jul;35(7):675-94. doi: 10.1038/hr.2012.77. Epub 2012 May 31.
6
High-resolution identity by descent mapping uncovers the genetic basis for blood pressure differences between spontaneously hypertensive rat lines.通过高分辨率同源性映射揭示自发性高血压大鼠品系间血压差异的遗传基础。
Circ Cardiovasc Genet. 2011 Jun;4(3):223-31. doi: 10.1161/CIRCGENETICS.110.958934. Epub 2011 Mar 15.
7
Protective importance of the myogenic response in the renal circulation.肌源性反应在肾循环中的保护作用。
Hypertension. 2009 Aug;54(2):393-8. doi: 10.1161/HYPERTENSIONAHA.109.133777. Epub 2009 Jun 22.
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RF-2 gene modulates proteinuria and albuminuria independently of changes in glomerular permeability in the fawn-hooded hypertensive rat.在黄褐毛高血压大鼠中，RF-2基因独立于肾小球通透性变化来调节蛋白尿和白蛋白尿。
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Br J Pharmacol. 1994 Jan;111(1):111-6. doi: 10.1111/j.1476-5381.1994.tb14031.x.
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