Asamura S, Hodes D S
Arch Virol. 1985;84(3-4):291-6. doi: 10.1007/BF01378982.
We investigated properties of the ts mutants that were selected during the course of persistent infection of Vero cells by parainfluenza virus type 3. The mutants demonstrated leakiness when infecting cells at high MOI and interfered with the growth of wild type virus, apparently by inhibiting a step prior to RNA synthesis.
我们研究了在副流感病毒3型持续感染Vero细胞过程中选择的温度敏感（ts）突变体的特性。这些突变体在高感染复数（MOI）感染细胞时表现出渗漏现象，并干扰野生型病毒的生长，显然是通过抑制RNA合成之前的一个步骤来实现的。
