Hanton G, Thiry E, Brochier B, Derboven G, Pastoret P P
Vet Microbiol. 1986 Feb;11(1-2):51-9. doi: 10.1016/0378-1135(86)90006-4.
Evolution of sensitizing antibodies involved in antibody-dependent cell-mediated cytotoxicity (ADCC), antibody-complement cell lysis and complement-facilitated ADCC was followed in bulls after primary infection by a wild strain of infectious bovine rhinotracheitis virus (bovine herpesvirus 1; BHV-1) and after experimental reactivation of the virus. These antibodies were detected between the 4th and the 7th day after primary infection, reached a maximum level after 2 weeks and rose slightly after reactivation of the virus following dexamethasone treatment. The presence of endogenous complement slightly enhanced the ADCC reaction.
在公牛初次感染传染性牛鼻气管炎病毒（牛疱疹病毒1型；BHV-1）野生株后以及病毒实验性再激活后，对参与抗体依赖性细胞介导的细胞毒性（ADCC）、抗体补体细胞裂解和补体促进的ADCC的致敏抗体的演变进行了跟踪研究。这些抗体在初次感染后的第4天至第7天被检测到，2周后达到最高水平，在用地塞米松治疗后病毒再激活后略有上升。内源性补体的存在略微增强了ADCC反应。
