Moser E, Roedler H D
Rofo. 1987 Jun;146(6):711-6. doi: 10.1055/s-2008-1048570.
Following the reactor accident at Chernobyl, this paper describes the biokinetics of radioiodine in man and discusses the radiation exposure resulting from intake of 131I. The risk of radiation-induced thyroid carcinomas and of congenital abnormalities is evaluated. Assuming a linear dose/risk relationship, one can calculate an increase in mortality from thyroid carcinomas amongst children in southern Germany of 100 to 101 per million children. For adults in southern Germany, and for the rest of the population in Germany, the figure is considerably lower. Gonadal dose from the 131I released is so small, compared with the annual natural radiation exposure, that it is not appropriate to discuss genetic effects.
在切尔诺贝利反应堆事故之后,本文描述了人体中放射性碘的生物动力学,并讨论了摄入131I所导致的辐射暴露。评估了辐射诱发甲状腺癌和先天性异常的风险。假设剂量/风险呈线性关系,可计算出德国南部儿童中因甲状腺癌导致的死亡率每百万儿童增加100至101例。对于德国南部的成年人以及德国其他人口而言,这一数字要低得多。与每年的天然辐射暴露相比,释放出的131I所产生的性腺剂量非常小,因此讨论遗传效应并不合适。