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本文引用的文献

1
Modulation of Cardiac Ventricular Excitability by GLP-1 (Glucagon-Like Peptide-1).GLP-1(胰高血糖素样肽-1)对心脏心室兴奋性的调制。
Circ Arrhythm Electrophysiol. 2018 Oct;11(10):e006740. doi: 10.1161/CIRCEP.118.006740.
2
Inconsistent relation of nonlinear heart rate variability indices to increasing vagal tone in healthy humans.健康人体非线性心率变异性指标与迷走神经张力增加的关系不一致。
Auton Neurosci. 2018 Sep;213:1-7. doi: 10.1016/j.autneu.2018.04.007. Epub 2018 May 1.
3
Thirty loci identified for heart rate response to exercise and recovery implicate autonomic nervous system.30 个与运动和恢复时心率反应相关的基因座,提示涉及自主神经系统。
Nat Commun. 2018 May 16;9(1):1947. doi: 10.1038/s41467-018-04148-1.
4
Genetic study links components of the autonomous nervous system to heart-rate profile during exercise.遗传研究将自主神经系统的组成部分与运动过程中的心率曲线联系起来。
Nat Commun. 2018 Mar 1;9(1):898. doi: 10.1038/s41467-018-03395-6.
5
Exercise therapy and autonomic function in heart failure patients: a systematic review and meta-analysis.运动疗法对心力衰竭患者自主神经功能的影响:系统评价和荟萃分析。
Heart Fail Rev. 2018 Jan;23(1):91-108. doi: 10.1007/s10741-017-9662-z.
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Exercise training and cardiac autonomic function in type 2 diabetes mellitus: A systematic review.2型糖尿病中的运动训练与心脏自主神经功能:一项系统综述。
Diabetes Metab Syndr. 2018 Jan-Mar;12(1):69-78. doi: 10.1016/j.dsx.2017.08.015. Epub 2017 Sep 6.
7
Autonomic regulation of systemic inflammation in humans: A multi-center, blinded observational cohort study.人体系统性炎症的自主调节:一项多中心、盲法观察性队列研究。
Brain Behav Immun. 2018 Jan;67:47-53. doi: 10.1016/j.bbi.2017.08.010. Epub 2017 Aug 12.
8
Vagal determinants of exercise capacity.迷走神经对运动能力的影响因素。
Nat Commun. 2017 May 18;8:15097. doi: 10.1038/ncomms15097.
9
Glucagon-like peptide-1 (GLP-1) mediates cardioprotection by remote ischaemic conditioning.胰高血糖素样肽-1(GLP-1)通过远程缺血预处理介导心脏保护作用。
Cardiovasc Res. 2016 Dec;112(3):669-676. doi: 10.1093/cvr/cvw216. Epub 2016 Oct 4.
10
Heritability of heart rate recovery and vagal rebound after exercise.运动后心率恢复及迷走神经反弹的遗传力
Eur J Appl Physiol. 2016 Dec;116(11-12):2167-2176. doi: 10.1007/s00421-016-3459-y. Epub 2016 Sep 10.

心脏迷走神经与运动。

Cardiac Vagus and Exercise.

机构信息

Centre for Cardiovascular and Metabolic Neuroscience, Neuroscience, Physiology and Pharmacology, University College London , London , United Kingdom.

William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London , London , United Kingdom.

出版信息

Physiology (Bethesda). 2019 Jan 1;34(1):71-80. doi: 10.1152/physiol.00041.2018.

DOI:10.1152/physiol.00041.2018
PMID:30540229
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6383634/
Abstract

Lower resting heart rate and high autonomic vagal activity are strongly associated with superior exercise capacity, maintenance of which is essential for general well-being and healthy aging. Recent evidence obtained in experimental studies using the latest advances in molecular neuroscience, combined with human exercise physiology, physiological modeling, and genomic data suggest that the strength of cardiac vagal activity causally determines our ability to exercise.

摘要

较低的静息心率和较高的自主迷走神经活动与优异的运动能力密切相关,而保持这种状态对于整体健康和健康衰老至关重要。最近在使用分子神经科学最新进展的实验研究中获得的证据,结合人类运动生理学、生理模型和基因组数据表明,心脏迷走神经活动的强度决定了我们进行运动的能力。