LiVolsi Virginia A, Baloch Zubair W
Department of Pathology and Laboratory Medicine, University of Pennsylvania Medical Center, Philadelphia, PA, United States.
Front Endocrinol (Lausanne). 2018 Dec 3;9:737. doi: 10.3389/fendo.2018.00737. eCollection 2018.
This article reviews those pathologic lesions which are associated with clinical and/or biochemical hyperthyroidism. Beginning with the descriptive pathology of classical Graves' disease and the less common toxic nodular goiter and hyper-functioning thyroid nodules, this paper describes the effects of non-thyroidal hormones, glandular function (including pituitary and hypothalamic lesions), ectopic production of thyroid stimulating proteins by non-thyroidal neoplasms, exogenous drug reactions causing hyper-function and finally conditions associated with a mechanic- destructive cause of hyperthyroidism.
本文回顾了那些与临床和/或生化甲状腺功能亢进相关的病理病变。本文从经典格雷夫斯病、较罕见的毒性结节性甲状腺肿和高功能甲状腺结节的描述性病理学开始，描述了非甲状腺激素的作用、腺体功能（包括垂体和下丘脑病变）、非甲状腺肿瘤异位产生甲状腺刺激蛋白、导致功能亢进的外源性药物反应，以及最后与机械性破坏导致甲状腺功能亢进相关的情况。
