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本文引用的文献
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Microglial Function Is Distinct in Different Anatomical Locations during Retinal Homeostasis and Degeneration.
Immunity. 2019 Mar 19;50(3):723-737.e7. doi: 10.1016/j.immuni.2019.02.007. Epub 2019 Mar 5.
2
Oxysterol Signatures Distinguish Age-Related Macular Degeneration from Physiologic Aging.
EBioMedicine. 2018 Jun;32:9-20. doi: 10.1016/j.ebiom.2018.05.035. Epub 2018 Jun 11.
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Macrophage microRNA-150 promotes pathological angiogenesis as seen in age-related macular degeneration.
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Subretinal mononuclear phagocytes induce cone segment loss via IL-1β.
Elife. 2016 Jul 20;5:e16490. doi: 10.7554/eLife.16490.
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Microglial phagocytosis and activation underlying photoreceptor degeneration is regulated by CX3CL1-CX3CR1 signaling in a mouse model of retinitis pigmentosa.
Glia. 2016 Sep;64(9):1479-91. doi: 10.1002/glia.23016. Epub 2016 Jun 17.
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Fate mapping reveals that microglia and recruited monocyte-derived macrophages are definitively distinguishable by phenotype in the retina.
Sci Rep. 2016 Feb 9;6:20636. doi: 10.1038/srep20636.
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Microglial phagocytosis of living photoreceptors contributes to inherited retinal degeneration.
EMBO Mol Med. 2015 Sep;7(9):1179-97. doi: 10.15252/emmm.201505298.
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CCR2(+) monocytes infiltrate atrophic lesions in age-related macular disease and mediate photoreceptor degeneration in experimental subretinal inflammation in Cx3cr1 deficient mice.
EMBO Mol Med. 2013 Nov;5(11):1775-93. doi: 10.1002/emmm.201302692. Epub 2013 Oct 21.
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Gene expression changes in aging retinal microglia: relationship to microglial support functions and regulation of activation.
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Senescence regulates macrophage activation and angiogenic fate at sites of tissue injury in mice.
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