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系统性红斑狼疮中髌腱的松弛和断裂。与继发性甲状旁腺功能亢进的关联。

Laxity and rupture of the patellar tendon in systemic lupus erythematosus. Association with secondary hyperparathyroidism.

作者信息

Babini S M, Arturi A, Marcos J C, Babini J C, Iniguez A M, Morteo O G

机构信息

Rheumatology Sections, Hospital General San Martin, La Plata, Argentina.

出版信息

J Rheumatol. 1988 Jul;15(7):1162-5.

PMID:3172117
Abstract

We describe a patient with systemic lupus erythematosus, chronic renal failure and secondary hyperparathyroidism who developed laxity and elongation of the patellar tendon, followed by its rupture one year later. Hydroxyapatite and urate crystals were identified from the chalky material surrounding the patellar tendon observed at surgery. These findings suggest that tendon laxity and elongation could possibly be detected before its rupture and that secondary hyperparathyroidism could be considered as another causative factor of tendon damage in patients with systemic lupus erythematosus.

摘要

我们描述了一名患有系统性红斑狼疮、慢性肾衰竭和继发性甲状旁腺功能亢进的患者,该患者出现了髌腱松弛和延长,一年后髌腱发生断裂。在手术中观察到围绕髌腱的灰白色物质中发现了羟基磷灰石和尿酸盐晶体。这些发现表明,在髌腱断裂前可能检测到其松弛和延长,并且继发性甲状旁腺功能亢进可被视为系统性红斑狼疮患者肌腱损伤的另一个致病因素。

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