NeuroV̇ASQ̇-Integrative Physiology Laboratory, Faculty of Physical Education, University of Brasília, Brasília, DF, Brazil.
Exerc Sport Sci Rev. 2020 Apr;48(2):83-91. doi: 10.1249/JES.0000000000000218.
The exercise pressor reflex (EPR) is engaged upon the activation of group III/IV skeletal muscle afferents and is one of the principal mediators of cardiovascular responses to exercise. This review explores the hypothesis that afferent signals from EPR communicate via GABAergic contacts within the brain stem to evoke parasympathetic withdrawal and sympathoexcitation to increase cardiac output, peripheral resistance, and blood pressure during exercise.
运动加压反射（EPR）是在 III/IV 群骨骼肌传入神经激活时产生的，是运动对心血管反应的主要介导者之一。本综述探讨了这样一种假设，即来自 EPR 的传入信号通过脑干中的 GABA 能接触进行通讯，从而引发副交感神经撤回和交感神经兴奋，以增加运动期间的心输出量、外周阻力和血压。
