The immunological basis of adverse drug reactions. A report on a symposium held in Liverpool on 6th April 1988.
作者信息
Park B K, Coleman J W
机构信息
Department of Pharmacology and Therapeutics, University of Liverpool.
出版信息
Br J Clin Pharmacol. 1988 Nov;26(5):491-5. doi: 10.1111/j.1365-2125.1988.tb05287.x.
Abstract
摘要
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本文引用的文献
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Oxidative metabolism of halothane in the production of altered hepatocyte membrane antigens in acute halothane-induced hepatic necrosis.氟烷在急性氟烷诱导的肝坏死中产生改变的肝细胞膜抗原过程中的氧化代谢。
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Predisposition to phenytoin hepatotoxicity assessed in vitro.体外评估苯妥英肝毒性易感性。
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T cell-dependent B-cell proliferation and activation induced by administration of the drug diphenylhydantoin to mice.
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Studies on the mechanism of drug sensitization: T-cell-dependent popliteal lymph node reaction to diphenylhydantoin.药物致敏机制的研究：T细胞依赖性对苯妥英的腘淋巴结反应。
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Drugs that induce systemic lupus erythematosus inhibit complement component C4.诱发系统性红斑狼疮的药物会抑制补体成分C4。
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Effect of storage of penicillin-G solutions on sensitisation to penicillin-G after intravenous administration.
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Hydralazine binds covalently to complement component C4. Different reactivity of C4A and C4B gene products.肼屈嗪与补体成分C4共价结合。C4A和C4B基因产物的不同反应性。
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Halothane hepatitis: a model of immune mediated drug hepatotoxicity.氟烷性肝炎：免疫介导的药物性肝毒性模型。
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