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[巩膜静脉压在继发性青光眼发病中的作用]

[The role of episcleral venous pressure in the development of secondary glaucomas].

作者信息

Jørgensen J S, Guthoff R

机构信息

Universitäts-Augenklinik, Rigshospitalet, Kopenhagen.

出版信息

Klin Monbl Augenheilkd. 1988 Nov;193(5):471-5. doi: 10.1055/s-2008-1050284.

DOI:10.1055/s-2008-1050284
PMID:3210642
Abstract

Between 1983 and 1987 the authors studied 64 patients with dilated episcleral vessels and open-angle glaucoma. Elevated episcleral venous pressure was found to be the cause of the increased intraocular pressure. Clinically, the cause of elevation of venous pressure included spontaneous carotid cavernous fistulas, Sturge-Weber syndrome, orbital tumors, endocrine exophthalmos, anterior scleritis and idiopathic cases. A variety of pathophysiological mechanisms causing elevated episcleral venous pressure were found. The clinical course and complications associated with elevated episcleral venous pressure are discussed.

摘要

1983年至1987年间,作者研究了64例巩膜表层血管扩张并伴有开角型青光眼的患者。发现巩膜静脉压升高是眼压升高的原因。临床上,静脉压升高的原因包括自发性颈内动脉海绵窦瘘、斯特奇-韦伯综合征、眼眶肿瘤、内分泌性突眼、前巩膜炎及特发性病例。发现了导致巩膜静脉压升高的多种病理生理机制。本文讨论了与巩膜静脉压升高相关的临床病程及并发症。

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