Takamatsu K, Takizawa T, Sato S, Sano A, Takahashi K, Murakami Y, Ota K
Department of Neurological Medicine, Ota Memorial Hospital, Hiroshima, Japan.
No Shinkei Geka. 1988 Nov;16(12):1383-7.
Two cases of acute idiopathic subdural hematoma with delayed intracranial hypertension were presented. The first case was a 68-year-old man admitted for vomiting following headache for eight days. There was no history of head trauma. A CT scan revealed a high-density mass that had a concave inner margin in the left temporo-parietal region with a slight midline shift. No vascular lesion was noted on the angiogram. The consciousness of the patient deteriorated suddenly on the 12th day. An operation was performed because of a marked midline shift on the CT. At operation, a subdural clot was removed. The postoperative recovery was good. The patient was discharged 7 days later without any neurological deficit. The second case was a 69-year-old man who was admitted with sudden onset of headache. There was no history of head trauma. A CT scan showed a high density mass in the right temporoparietal subdural space with a slight midline shift. The consciousness of the patient deteriorated suddenly on the 15th day. An operation was performed because of a marked midline shift on the CT. At operation, a subdural hematoma was removed. Two days later, suddenly his consciousness deteriorated. A CT scan showed his severe brain edema with a marked midline shift without increased hematoma. External decompression was performed immediately. The postoperative recovery was very good and 40 days after the second operation, the patient was discharged with no neurological deficit. The delayed intracranial hypertension appeared in these two cases about 10 days after the initial symptom. Two kinds of mechanisms are suspected: 1) swelling of the hematoma because of the adsorption of cerebrospinal fluid, 2) the occurrence of secondary brain edema. From our experience, a repeated CT scan is necessary for 2 to 3 weeks.(ABSTRACT TRUNCATED AT 250 WORDS)
本文报告了两例急性特发性硬膜下血肿伴迟发性颅内高压的病例。第一例为一名68岁男性,因头痛八天后呕吐入院。无头部外伤史。CT扫描显示左颞顶叶区域有一高密度肿块,内缘呈凹形,中线稍有移位。血管造影未发现血管病变。患者在第12天意识突然恶化。由于CT显示中线明显移位,遂行手术。术中清除了硬膜下血块。术后恢复良好。患者7天后出院,无任何神经功能缺损。第二例为一名69岁男性,因突发头痛入院。无头部外伤史。CT扫描显示右侧颞顶叶硬膜下间隙有一高密度肿块,中线稍有移位。患者在第15天意识突然恶化。由于CT显示中线明显移位,遂行手术。术中清除了硬膜下血肿。两天后,他的意识突然恶化。CT扫描显示严重脑水肿,中线明显移位,血肿未增加。立即进行了外减压术。术后恢复非常好,第二次手术后40天,患者出院,无神经功能缺损。这两例迟发性颅内高压均在初始症状出现约10天后出现。怀疑有两种机制:1)血肿因脑脊液吸附而肿胀,2)继发性脑水肿的发生。根据我们的经验,术后2至3周需要重复进行CT扫描。(摘要截取自250字)