Sielaff S
Universitätsklinik für Innere Medizin Theodor Brugsch, Humboldt-Universität zu Berlin.
Dtsch Z Verdau Stoffwechselkr. 1988;48(3-4):162-72.
After ingestion of a solid test meal the postprandial motor activity in 17 dyspeptic patients and 12 healthy controls were examined. In all individuals the gastric emptying was measured by scintigraphy. - The antral pressure activity after food intake was delayed in dyspepsia and showed a distinct reduction with time (antral hypomotility). In contrast the postprandial duodenal motility was increased significantly (duodenal hyperdyskinesia). All 6 dyspeptic patients with prolonged gastric emptying had gastroduodenal manometric abnormalities. - Our results suggest that in chronic dyspepsia the interdigestive and postprandial motility is often disturbed. The delayed gastric emptying occurs because of impaired antral peristalsis and/or increase of duodenal resistance.
在摄入固体试验餐后，对17名消化不良患者和12名健康对照者的餐后运动活性进行了检查。通过闪烁扫描法测量了所有个体的胃排空情况。——消化不良患者进食后胃窦压力活性延迟，且随时间明显降低（胃窦动力不足）。相比之下，餐后十二指肠动力显著增加（十二指肠运动功能亢进）。所有6名胃排空延长的消化不良患者均有胃十二指肠测压异常。——我们的结果表明，在慢性消化不良中，消化间期和餐后运动常受到干扰。胃排空延迟是由于胃窦蠕动受损和/或十二指肠阻力增加所致。
