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以脑性盐耗综合征为表现的神经型布鲁氏菌病

Neurobrucellosis Presented by Cerebral Salt Wasting.

作者信息

Kemec Zeki, Tüzün Cevat, Yıldırım İdris

机构信息

Nephrology, Batman District State Hospital, Batman, TUR.

Radiology, Batman District State Hospital, Batman, TUR.

出版信息

Cureus. 2020 May 25;12(5):e8275. doi: 10.7759/cureus.8275.

Abstract

This case reported here was a 27-year-old female patient, and she had no chronic disease other than hypothyroidism. She was brought to the ER with complaints of fever, chills, weakness, and hyponatremia. She has been diagnosed with brucellosis using a serum tube agglutination test (STAT). Cerebrospinal fluid (CSF) analysis and brain MRI findings supported the central nervous system (CNS) involvement of the brucella. Despite intense 3% NaCl hydration, her hyponatremia was persisting. Sodium stabilized on the 14th day when the antibrucellosis treatment effect was settled. Hyponatremia was attributed to cerebral salt wasting (CSW) due to neurobrucellosis.

摘要

此处报告的病例是一名27岁女性患者,除甲状腺功能减退外无其他慢性疾病。她因发热、寒战、虚弱和低钠血症被送往急诊室。通过血清试管凝集试验(STAT)诊断为布鲁氏菌病。脑脊液(CSF)分析和脑部MRI检查结果支持布鲁氏菌累及中枢神经系统(CNS)。尽管进行了强化的3%氯化钠补液,她的低钠血症仍持续存在。在抗布鲁氏菌病治疗效果显现后的第14天,血钠稳定下来。低钠血症归因于神经型布鲁氏菌病导致的脑性盐耗竭(CSW)。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/33e0/7314369/f007b97ba074/cureus-0012-00000008275-i01.jpg

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