• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

相似文献

1
Corrigendum to "Macrophage Migration Inhibitory Factor Promotes the Interaction between the Tumor, Macrophages, and T Cells to Regulate the Progression of Chemically Induced Colitis-Associated Colorectal Cancer".《巨噬细胞迁移抑制因子促进肿瘤、巨噬细胞和T细胞之间的相互作用以调节化学诱导的结肠炎相关结直肠癌进展》的勘误
Mediators Inflamm. 2020 Jun 17;2020:2195341. doi: 10.1155/2020/2195341. eCollection 2020.
2
Macrophage Migration Inhibitory Factor Promotes the Interaction between the Tumor, Macrophages, and T Cells to Regulate the Progression of Chemically Induced Colitis-Associated Colorectal Cancer.巨噬细胞移动抑制因子促进肿瘤、巨噬细胞和 T 细胞的相互作用,调节化学诱导的结肠炎相关结直肠癌的进展。
Mediators Inflamm. 2019 Jul 10;2019:2056085. doi: 10.1155/2019/2056085. eCollection 2019.
3
Ninjurin1 inhibits colitis-mediated colon cancer development and growth by suppression of macrophage infiltration through repression of FAK signaling.神经损伤诱导蛋白1通过抑制黏着斑激酶信号通路来抑制巨噬细胞浸润,从而抑制结肠炎介导的结肠癌的发生和生长。
Oncotarget. 2016 May 17;7(20):29592-604. doi: 10.18632/oncotarget.9020.
4
M2 Macrophage-Derived Exosomes Promote Cell Migration and Invasion in Colon Cancer.M2 巨噬细胞衍生的外泌体促进结肠癌中的细胞迁移和侵袭。
Cancer Res. 2019 Jan 1;79(1):146-158. doi: 10.1158/0008-5472.CAN-18-0014. Epub 2018 Nov 6.
5
Obesity exacerbates colitis-associated cancer via IL-6-regulated macrophage polarisation and CCL-20/CCR-6-mediated lymphocyte recruitment.肥胖通过 IL-6 调节的巨噬细胞极化和 CCL-20/CCR-6 介导的淋巴细胞募集加剧结肠炎相关癌症。
Nat Commun. 2018 Apr 25;9(1):1646. doi: 10.1038/s41467-018-03773-0.
6
Wuchereria bancrofti macrophage migration inhibitory factor-2 (rWbaMIF-2) ameliorates experimental colitis.班氏丝虫巨噬细胞移动抑制因子-2(rWbaMIF-2)可改善实验性结肠炎。
Parasite Immunol. 2020 Apr;42(4):e12698. doi: 10.1111/pim.12698. Epub 2020 Feb 6.
7
Erratum: [Corrigendum] miR‑590‑5p may regulate colorectal cancer cell viability and migration by targeting PDCD4.勘误:[更正] miR-590-5p可能通过靶向PDCD4调控结肠癌细胞的活力和迁移。
Exp Ther Med. 2024 Jan 17;27(3):104. doi: 10.3892/etm.2024.12392. eCollection 2024 Mar.
8
Erratum: ACAT2 Promotes Cell Proliferation and Associates with Malignant Progression in Colorectal Cancer [Corrigendum].勘误:ACAT2促进细胞增殖并与结直肠癌的恶性进展相关[勘误]。
Onco Targets Ther. 2021 Aug 31;14:4641-4643. doi: 10.2147/OTT.S336190. eCollection 2021.
9
Specific tumor-derived CCL2 mediated by pyruvate kinase M2 in colorectal cancer cells contributes to macrophage recruitment in tumor microenvironment.由结肠癌细胞中丙酮酸激酶M2介导的特定肿瘤源性CCL2有助于肿瘤微环境中巨噬细胞的募集。
Tumour Biol. 2017 Mar;39(3):1010428317695962. doi: 10.1177/1010428317695962.
10
Corrigendum to "PIAS1-FAK Interaction Promotes the Survival and Progression of Non-Small Cell Lung Cancer" [ 18 (2016) 282-293].《PIAS1与FAK的相互作用促进非小细胞肺癌的存活与进展》[18(2016)282 - 293]的勘误
Neoplasia. 2016 Jul 17;18(7):457. doi: 10.1016/j.neo.2016.06.005. eCollection 2016 Jul.

引用本文的文献

1
Colorectal cancer prognosis: insights from the tumor immune microenvironment and gut microbiota.结直肠癌预后:来自肿瘤免疫微环境和肠道微生物群的见解
J Gastrointest Oncol. 2025 Aug 30;16(4):1521-1533. doi: 10.21037/jgo-2025-517. Epub 2025 Aug 27.
2
Hsp90-stabilized MIF supports tumor progression via macrophage recruitment and angiogenesis in colorectal cancer.Hsp90 稳定化的 MIF 通过招募巨噬细胞和血管生成促进结直肠癌的进展。
Cell Death Dis. 2021 Feb 4;12(2):155. doi: 10.1038/s41419-021-03426-z.

本文引用的文献

1
Macrophage Migration Inhibitory Factor Promotes the Interaction between the Tumor, Macrophages, and T Cells to Regulate the Progression of Chemically Induced Colitis-Associated Colorectal Cancer.巨噬细胞移动抑制因子促进肿瘤、巨噬细胞和 T 细胞的相互作用,调节化学诱导的结肠炎相关结直肠癌的进展。
Mediators Inflamm. 2019 Jul 10;2019:2056085. doi: 10.1155/2019/2056085. eCollection 2019.

《巨噬细胞迁移抑制因子促进肿瘤、巨噬细胞和T细胞之间的相互作用以调节化学诱导的结肠炎相关结直肠癌进展》的勘误

Corrigendum to "Macrophage Migration Inhibitory Factor Promotes the Interaction between the Tumor, Macrophages, and T Cells to Regulate the Progression of Chemically Induced Colitis-Associated Colorectal Cancer".

作者信息

Pacheco-Fernández Thalia, Juárez-Avelar Imelda, Illescas Oscar, Terrazas Luis I, Hernández-Pando Rogelio, Pérez-Plasencia Carlos, Gutiérrez-Cirlos Emma B, Ávila-Moreno Federico, Chirino Yolanda I, Reyes José Luis, Maldonado Vilma, Rodriguez-Sosa Miriam

机构信息

Biomedicine Unit, Facultad de Estudios Superiores Iztacala, Universidad Nacional Autónoma de México (UNAM), Tlalnepantla, C.P, 54090, Mexico.

Experimental Pathology Section, National Institute of Medical Sciences and Nutrition "Salvador Zubirán", Tlalpan, C.P., 14000 Mexico City, Mexico.

出版信息

Mediators Inflamm. 2020 Jun 17;2020:2195341. doi: 10.1155/2020/2195341. eCollection 2020.

DOI:10.1155/2020/2195341
PMID:32655310
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7317330/
Abstract

[This corrects the article DOI: 10.1155/2019/2056085.].

摘要

[本文更正了文章的数字对象标识符:10.1155/2019/2056085。]