Suzuki J B
Department of Periodontics, University of Maryland, Baltimore.
Dent Clin North Am. 1988 Apr;32(2):195-216.
A classification for gingivitis and periodontitis has been proposed based on clinical observations and immunologic parameters (summarized in Table 10). Overlapping clinical situations and exceptions to the rule certainly exist and pose challenges to clinicians during diagnosis, prognosis, and treatment planning. The clinical significance of making the diagnosis of either rapidly progressive periodontitis, juvenile, or prepubertal forms of periodontitis compared with adult periodontitis must be considered. A diagnosis of early-onset disease may (1) modify the periodontal treatment plan and may include the use of systemic antibiotics (for example, tetracyclines) or antiplaque agents (for example, sanguinarine, chlorhexidine); (2) modify the prosthetic treatment plan and require longer periods of monitoring before extensive replacement or splinting is initiated; (3) increase the frequency of periodontal recall appointments (at least four to six times during the first year or two following peridontal therapy); (4) implicate a genetic basis for the disease with the possibility of peridontal involvement of other family members; (5) influence decisions on prognosis; and (6) alert the clinician for potential future rapid periodontal breakdown. Future studies will further define and characterize all forms of periodontal diseases. Clinical, microbial, and immunologic information may permit inclusion of the relatively rare forms of periodontitis such as ANUG periodontitis and refractory periodontitis. Understanding the underlying biologic mechanisms of the pathogenesis of the periodontal diseases, coupled with clinical observations, will permit improved clinical investigations that ultimately will improve therapeutic approaches.
基于临床观察和免疫参数,已提出了一种牙龈炎和牙周炎的分类方法(总结于表10)。重叠的临床情况和该规则的例外情况确实存在,在诊断、预后和治疗计划制定过程中给临床医生带来了挑战。必须考虑诊断快速进展性牙周炎、青少年或青春期前牙周炎与成人牙周炎相比的临床意义。早期发病疾病的诊断可能会:(1)修改牙周治疗计划,可能包括使用全身抗生素(如四环素)或抗牙菌斑药物(如血根碱、氯己定);(2)修改修复治疗计划,并在开始广泛修复或夹板固定之前需要更长时间的监测;(3)增加牙周复查预约的频率(在牙周治疗后的头一两年内至少四至六次);(4)暗示该疾病有遗传基础,其他家庭成员可能有牙周受累的可能性;(5)影响预后的判断;(6)提醒临床医生注意未来可能出现的快速牙周破坏。未来的研究将进一步明确和描述所有形式的牙周疾病。临床、微生物和免疫信息可能有助于纳入相对罕见的牙周炎形式,如急性坏死性溃疡性龈炎牙周炎和难治性牙周炎。了解牙周疾病发病机制的潜在生物学机制,并结合临床观察,将有助于改进临床研究,最终改善治疗方法。