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肝硬化腹水合并获得性免疫缺陷综合征患者自发性B群肠炎沙门氏菌腹膜炎

Spontaneous group B Salmonella enteritidis peritonitis in cirrhotic ascites and acquired immune deficiency syndrome.

作者信息

Reddy K R, Chan J C, Smiley D, Jeffers L J, Schiff E R

机构信息

Department of Medicine, Veterans Administration Medical Center, University of Miami School of Medicine, Florida.

出版信息

Am J Gastroenterol. 1988 Aug;83(8):882-4.

PMID:3293435
Abstract

Salmonella represents an infrequent cause of spontaneous bacterial peritonitis in cirrhotic ascites. A cirrhotic patient, an alcoholic and former intravenous drug abuser, developed spontaneous group B Salmonella enteritidis (undetermined serotype) bacterial peritonitis. The isolation of this pathogen, a common cause of bacteremia in acquired immune deficiency syndrome, led to the serological determination of infection with human immune deficiency virus (HIV).

摘要

沙门氏菌是肝硬化腹水患者自发性细菌性腹膜炎的罕见病因。一名肝硬化患者,有酗酒史且曾静脉注射吸毒,发生了自发性B群肠炎沙门氏菌(血清型未确定)细菌性腹膜炎。这种病原体是获得性免疫缺陷综合征中菌血症的常见病因,其分离培养导致了对人类免疫缺陷病毒(HIV)感染的血清学检测。

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Spontaneous group B Salmonella enteritidis peritonitis in cirrhotic ascites and acquired immune deficiency syndrome.肝硬化腹水合并获得性免疫缺陷综合征患者自发性B群肠炎沙门氏菌腹膜炎
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Current Status and Prospects of Spontaneous Peritonitis in Patients with Cirrhosis.肝硬化自发性腹膜炎的现状与展望。
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Non-typhoidal salmonella: an unusual cause of spontaneous bacterial peritonitis in decompensated cirrhosis.
非伤寒沙门氏菌:失代偿性肝硬化自发性细菌性腹膜炎的一个不常见病因。
Gastroenterol Rep (Oxf). 2014 Aug;2(3):242-4. doi: 10.1093/gastro/gou018. Epub 2014 Mar 19.
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Spontaneous bacterial peritonitis from Salmonella: an unusual bacterium with unusual presentation.自发性细菌性腹膜炎由沙门氏菌引起:一种不寻常的细菌引起的不寻常表现。
Hepatol Int. 2008 Sep;2(3):388-9. doi: 10.1007/s12072-008-9087-9. Epub 2008 Jul 25.
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Spontaneous non-typhoidal Salmonella peritonitis in patients with serious underlying disorders.患有严重基础疾病患者的自发性非伤寒沙门氏菌腹膜炎
Infection. 1999 May-Jun;27(3):224-7. doi: 10.1007/BF02561536.
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Surgery and Salmonella.手术与沙门氏菌
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Salmonella enteritidis empyema complicating lupus pleuritis.肠炎沙门氏菌脓胸并发狼疮性胸膜炎。
Postgrad Med J. 1991 Aug;67(790):778-9. doi: 10.1136/pgmj.67.790.778-a.