Regulation of sugar utilization in Saccharomyces species.
作者信息
Carlson M
机构信息
Department of Genetics, College of Physicians and Surgeons, Columbia University, New York, New York 10032.
出版信息
J Bacteriol. 1987 Nov;169(11):4873-7. doi: 10.1128/jb.169.11.4873-4877.1987.
Abstract
摘要
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本文引用的文献
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Pleiotropic properties of a yeast mutant insensitive to catabolite repression.酵母突变体对分解代谢物阻遏不敏感的多效性特性。
Genetics. 1980 Apr;94(4):921-8. doi: 10.1093/genetics/94.4.921.
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Pleiotropic Mutations at the TUP1 Locus That Affect the Expression of Mating-Type-Dependent Functions in SACCHAROMYCES CEREVISIAE.TUP1 基因座的多效突变影响酿酒酵母交配型依赖性功能的表达。
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Mutants of yeast defective in sucrose utilization.蔗糖利用存在缺陷的酵母突变体。
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4
Genetic and biochemical evidence for hexokinase PII as a key enzyme involved in carbon catabolite repression in yeast.己糖激酶PII作为参与酵母中碳分解代谢物阻遏的关键酶的遗传和生化证据。
Mol Gen Genet. 1980;178(3):633-7. doi: 10.1007/BF00337871.
5
Glycolytic enzymes and intermediates in carbon catabolite repression mutants of Saccharomyces cerevisiae.酿酒酵母碳代谢物阻遏突变体中的糖酵解酶和中间产物。
Mol Gen Genet. 1980 Jan;177(2):345-50. doi: 10.1007/BF00267449.
6
Organization of the SUC gene family in Saccharomyces.酿酒酵母中SUC基因家族的组织方式。
Mol Cell Biol. 1983 Mar;3(3):351-9. doi: 10.1128/mcb.3.3.351-359.1983.
7
Isolation and characterization of dominant mutations resistant to carbon catabolite repression of galactokinase synthesis in Saccharomyces cerevisiae.酿酒酵母中对半乳糖激酶合成的碳代谢物阻遏具有抗性的显性突变的分离与鉴定
Mol Cell Biol. 1981 Feb;1(2):83-93. doi: 10.1128/mcb.1.2.83-93.1981.
8
A GAL10-CYC1 hybrid yeast promoter identifies the GAL4 regulatory region as an upstream site.一个GAL10-CYC1杂交酵母启动子将GAL4调控区域鉴定为一个上游位点。
Proc Natl Acad Sci U S A. 1982 Dec;79(23):7410-4. doi: 10.1073/pnas.79.23.7410.
9
Molecular cloning of the GAL80 gene from Saccharomyces cerevisiae and characterization of a gal80 deletion.来自酿酒酵母的GAL80基因的分子克隆及gal80缺失的表征
Gene. 1984 Dec;32(1-2):75-82. doi: 10.1016/0378-1119(84)90034-9.
10
Nucleotide sequence of the yeast regulatory gene GAL80.酵母调控基因GAL80的核苷酸序列。
Nucleic Acids Res. 1984 Dec 21;12(24):9287-98. doi: 10.1093/nar/12.24.9287.
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