Enhanced expression of angiotensin-converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon-γ: implication of inflammatory milieu in skin tropism of SARS-CoV-2.
作者信息
Tembhre M K, Parihar A S, Sharma V K, Imran S, Bhari N, Lakshmy R, Bhalla A
机构信息
Department of Dermatology and Venereology, AIIMS, New Delhi, India.
Department of Cardiac Biochemistry, AIIMS, New Delhi, India.
出版信息
Br J Dermatol. 2021 Mar;184(3):577-579. doi: 10.1111/bjd.19670. Epub 2020 Dec 28.
Abstract
摘要
相似文献
1
Enhanced expression of angiotensin-converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon-γ: implication of inflammatory milieu in skin tropism of SARS-CoV-2.血管紧张素转换酶2在银屑病皮肤中的表达增强及其在角质形成细胞中被干扰素-γ上调：炎症环境在SARS-CoV-2皮肤嗜性中的作用
Br J Dermatol. 2021 Mar;184(3):577-579. doi: 10.1111/bjd.19670. Epub 2020 Dec 28.
2
Response to Tembhre et al.: 'Enhanced expression of angiotensin-converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon-γ: implication of inflammatory milieu in skin tropism of SARS-CoV-2'.回复 Tembhre 等人：“银屑病皮肤中血管紧张素转换酶 2 的表达增强及其被干扰素-γ上调：炎症微环境在 SARS-CoV-2 皮肤嗜性中的作用”。
Br J Dermatol. 2021 May;184(5):984. doi: 10.1111/bjd.19714. Epub 2021 Feb 1.
3
Expression of ACE2 and TMPRSS2 Proteins in the Upper and Lower Aerodigestive Tracts of Rats: Implications on COVID 19 Infections.大鼠上下呼吸道中 ACE2 和 TMPRSS2 蛋白的表达：对 COVID-19 感染的影响。
Laryngoscope. 2021 Mar;131(3):E932-E939. doi: 10.1002/lary.29132. Epub 2020 Oct 19.
4
Increased expression of key SARS-CoV-2 entry points in multiple tissues in individuals with NAFLD.非酒精性脂肪性肝病（NAFLD）患者多种组织中新冠病毒（SARS-CoV-2）关键进入点的表达增加。
J Hepatol. 2021 Mar;74(3):748-749. doi: 10.1016/j.jhep.2020.12.007. Epub 2020 Dec 16.
5
Bromelain inhibits SARS-CoV-2 infection via targeting ACE-2, TMPRSS2, and spike protein.菠萝蛋白酶通过靶向血管紧张素转换酶2（ACE-2）、跨膜丝氨酸蛋白酶2（TMPRSS2）和刺突蛋白来抑制新型冠状病毒2（SARS-CoV-2）感染。
Clin Transl Med. 2021 Feb;11(2):e281. doi: 10.1002/ctm2.281.
6
Priming of SARS-CoV-2 S protein by several membrane-bound serine proteinases could explain enhanced viral infectivity and systemic COVID-19 infection.几种膜结合丝氨酸蛋白酶对 SARS-CoV-2 S 蛋白的启动作用可以解释增强的病毒感染力和全身性 COVID-19 感染。
J Biol Chem. 2021 Jan-Jun;296:100135. doi: 10.1074/jbc.REV120.015980. Epub 2020 Dec 6.
7
TMPRSS11D and TMPRSS13 Activate the SARS-CoV-2 Spike Protein.TMPRSS11D 和 TMPRSS13 激活 SARS-CoV-2 刺突蛋白。
Viruses. 2021 Feb 28;13(3):384. doi: 10.3390/v13030384.
8
Omicron's molecular structure could help explain its global takeover.奥密克戎的分子结构有助于解释它在全球的传播态势。
Nature. 2022 Feb;602(7897):373-374. doi: 10.1038/d41586-022-00292-3.
9
COVID-19 in men: With or without virus in semen, spermatogenesis may be impaired.男性的新冠病毒感染：无论精液中有无病毒，精子发生都可能受损。
Andrologia. 2021 Feb;53(1):e13878. doi: 10.1111/and.13878. Epub 2020 Oct 30.
10
Distinctive Roles of Furin and TMPRSS2 in SARS-CoV-2 Infectivity.弗林蛋白酶和 TMPRSS2 在 SARS-CoV-2 感染中的独特作用。
J Virol. 2022 Apr 27;96(8):e0012822. doi: 10.1128/jvi.00128-22. Epub 2022 Mar 28.
引用本文的文献
1
COVID-19 Among Patients with Psoriasis: A Single-Center Retrospective Cross-Sectional Study.银屑病患者中的新型冠状病毒肺炎：一项单中心回顾性横断面研究。
Infect Dis Clin Microbiol. 2023 Jun 23;5(2):127-135. doi: 10.36519/idcm.2023.192. eCollection 2023 Jun.
2
The Renin-Angiotensin System: The Challenge behind Autoimmune Dermatological Diseases.肾素-血管紧张素系统：自身免疫性皮肤病背后的挑战
Diagnostics (Basel). 2023 Nov 7;13(22):3398. doi: 10.3390/diagnostics13223398.
3
Receptors and Cofactors That Contribute to SARS-CoV-2 Entry: Can Skin Be an Alternative Route of Entry?有助于 SARS-CoV-2 进入的受体和辅助因子：皮肤是否可能成为另一种进入途径？
Int J Mol Sci. 2023 Mar 26;24(7):6253. doi: 10.3390/ijms24076253.
4
SARS-CoV-2 and Skin: New Insights and Perspectives.SARS-CoV-2 与皮肤：新的见解和观点。
Biomolecules. 2022 Aug 31;12(9):1212. doi: 10.3390/biom12091212.
5
IL-33 Enhances ACE2 Expression on Epidermal Keratinocytes in Atopic Dermatitis: A Plausible Issue for SARS-CoV-2 Transmission in Inflamed Atopic Skin.白细胞介素-33增强特应性皮炎患者表皮角质形成细胞上血管紧张素转换酶2的表达：炎症性特应性皮肤中新型冠状病毒传播的一个可能问题。
Biomedicines. 2022 May 20;10(5):1183. doi: 10.3390/biomedicines10051183.
6
might transmit through the skin while the skin barrier function could be the mediator.可能通过皮肤传播，而皮肤屏障功能可能是介导因素。
Med Hypotheses. 2022 Feb;159:110752. doi: 10.1016/j.mehy.2021.110752. Epub 2022 Jan 3.
7
Biologics for Psoriasis During the COVID-19 Pandemic.新冠疫情期间用于治疗银屑病的生物制剂
Front Med (Lausanne). 2021 Dec 6;8:759568. doi: 10.3389/fmed.2021.759568. eCollection 2021.
8
Skin Aging, Cellular Senescence and Natural Polyphenols.皮肤衰老、细胞衰老与天然多酚
Int J Mol Sci. 2021 Nov 23;22(23):12641. doi: 10.3390/ijms222312641.
9
Multifunctional angiotensin converting enzyme 2, the SARS-CoV-2 entry receptor, and critical appraisal of its role in acute lung injury.多功能血管紧张素转换酶 2，SARS-CoV-2 进入受体，以及对其在急性肺损伤中作用的批判性评价。
Biomed Pharmacother. 2021 Apr;136:111193. doi: 10.1016/j.biopha.2020.111193. Epub 2021 Jan 5.
10
Response to Tembhre et al.: 'Enhanced expression of angiotensin-converting enzyme 2 in psoriatic skin and its upregulation in keratinocytes by interferon-γ: implication of inflammatory milieu in skin tropism of SARS-CoV-2'.回复 Tembhre 等人：“银屑病皮肤中血管紧张素转换酶 2 的表达增强及其被干扰素-γ上调：炎症微环境在 SARS-CoV-2 皮肤嗜性中的作用”。
Br J Dermatol. 2021 May;184(5):984. doi: 10.1111/bjd.19714. Epub 2021 Feb 1.
本文引用的文献
1
Lack of Evidence for an Increased Risk of Severe COVID-19 in Psoriasis Patients on Biologics: A Cohort Study from Northeast Italy.生物制剂治疗的银屑病患者发生重症 COVID-19 风险增加缺乏证据：来自意大利东北部的一项队列研究
Am J Clin Dermatol. 2020 Oct;21(5):749-751. doi: 10.1007/s40257-020-00552-w.
2
Cutaneous manifestations of SARS-CoV-2 infection: a clinical update.新型冠状病毒感染的皮肤表现：临床最新进展。
J Eur Acad Dermatol Venereol. 2020 Nov;34(11):2499-2504. doi: 10.1111/jdv.16774. Epub 2020 Jul 20.
3
SARS-CoV-2 Receptor ACE2 Is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Detected in Specific Cell Subsets across Tissues.SARS-CoV-2 受体 ACE2 是人类气道上皮细胞中的一种干扰素刺激基因，可在组织中的特定细胞亚群中检测到。
Cell. 2020 May 28;181(5):1016-1035.e19. doi: 10.1016/j.cell.2020.04.035. Epub 2020 Apr 27.
4
Multiorgan and Renal Tropism of SARS-CoV-2.新型冠状病毒2019（SARS-CoV-2）的多器官及肾脏嗜性
N Engl J Med. 2020 Aug 6;383(6):590-592. doi: 10.1056/NEJMc2011400. Epub 2020 May 13.
5
SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor.严重急性呼吸综合征冠状病毒 2 型（SARS-CoV-2）进入细胞依赖于 ACE2 和 TMPRSS2，可被一种临床验证的蛋白酶抑制剂所阻断。
Cell. 2020 Apr 16;181(2):271-280.e8. doi: 10.1016/j.cell.2020.02.052. Epub 2020 Mar 5.
6
The distinct role and regulatory mechanism of IL-17 and IFN-γ in the initiation and development of plaque vs guttate psoriasis.IL-17 和 IFN-γ 在斑块型银屑病与点滴状银屑病的发生和发展中的独特作用和调控机制。
J Dermatol Sci. 2018 Oct;92(1):106-113. doi: 10.1016/j.jdermsci.2018.07.001. Epub 2018 Jul 7.
7
Participation of T cell immunoglobulin and mucin domain-3 (TIM-3) and its ligand (galectin-9) in the pathogenesis of active generalized vitiligo.T细胞免疫球蛋白黏蛋白结构域3（TIM-3）及其配体（半乳糖凝集素-9）在活动性泛发性白癜风发病机制中的作用
Immunol Res. 2015 May;62(1):23-34. doi: 10.1007/s12026-015-8632-6.
8
Alteration in regulatory T cells and programmed cell death 1-expressing regulatory T cells in active generalized vitiligo and their clinical correlation.活动期泛发性白癜风患者调节性 T 细胞及程序性死亡受体 1 表达的调节性 T 细胞的改变及其临床相关性。
Br J Dermatol. 2015 Apr;172(4):940-50. doi: 10.1111/bjd.13511. Epub 2015 Feb 27.
Zotero 插件