[通过对抗刺激的研究探索控制炎症反应的内源性机制：前列腺素的释放、激肽的形成和白细胞的聚集]
[Exploration of endogenous mechanisms controlling the inflammatory reaction, by the study of counter-irritation: release of prostaglandins, formation of kinins and accumulation of leukocytes].
作者信息
Damas J, Deflandre E
机构信息
Institut Léon-Fredericq, Physiologie humaine normale et pathologique, Liege, Belgique.
出版信息
Pathol Biol (Paris). 1987 Nov;35(9):1253-62.
PMID:3320903
Abstract
This review was focused on counter irritation by turpentine in the rat. The involvement of sympathetic system, adrenal steroids, beta-endorphin, axon reflexes, haptoglobin, alpha-2-macroglobulin, complement activation, prostaglandin synthesis, kinin formation and leukocyte accumulation was considered as possible explanation of the anti-inflammatory effect of counter irritation by turpentine. The main mechanism consists of the reduction of leucocyte accumulation.
摘要
本综述聚焦于松节油在大鼠中的抗刺激作用。交感神经系统、肾上腺类固醇、β-内啡肽、轴突反射、触珠蛋白、α-2-巨球蛋白、补体激活、前列腺素合成、激肽形成和白细胞聚集的参与被认为是松节油抗刺激抗炎作用的可能解释。主要机制包括白细胞聚集的减少。
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