Piatt Joseph
Division of Neurosurgery, Nemours / A I duPont Hospital for Children, 1600 Rockland Road, Wilmington, DE, 19803, USA.
Departments of Neurological Surgery and Pediatrics, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, PA, USA.
Inj Epidemiol. 2021 Jan 11;8(1):1. doi: 10.1186/s40621-020-00295-6.
In the United States social disparities in health outcomes are found wherever they are sought, and they have been documented extensively in trauma care. Because social factors cannot cause a trauma outcome directly, there must exist mediating causal factors related to the nature and severity of the injury, the robustness of the victim, access to care, or processes of care. An understanding these mediators is the point of departure for addressing inequities in outcomes.
Data were extracted from the registry of the Trauma Quality Improvement Program of the American College of Surgeons for 2007 through 2010. Inclusion criteria were age less than 19 years and head Abbreviated Injury Scale score of 4, 5, or 6. An Oaxaca-Blinder decomposition was undertaken to analyze the relative contributions of a large set of covariates to the difference in mortality rates between Black and White children. Covariates were aggregated into the following categories: "Severity," "Structure and Process," "Mechanism," "Demographics," and "Insurance." Eligible for analysis were 7273 White children and 2320 Black children. There were 1661 deaths (17.3%) The raw mortality rates were 15.6 and 22.8% for White and Black children, respectively. Factors categorized as "Severity" accounted for 95% of the mortality difference, "Mechanism" accounted for 13%, "Insurance" accounted for 5%, and "Demographics" accounted for 2%. The contribution of "Structure and Process" did not attain statistical significance.
Severity of injury accounts for most of the disparity between Black and White children in traumatic brain injury mortality rates. Mechanism, insurance status, and gender make a small contributions. Because insurance status like other social factors cannot directly affect trauma survival, what mediates its contribution requires further study.
在美国,无论在何处探寻,都能发现健康结果方面的社会差异,并且这些差异在创伤护理领域已有大量记录。由于社会因素无法直接导致创伤结果,所以必然存在与损伤的性质和严重程度、受害者的健康状况、获得医疗服务的机会或医疗过程相关的中介因果因素。了解这些中介因素是解决结果不平等问题的出发点。
数据取自美国外科医师学会创伤质量改进项目2007年至2010年的登记册。纳入标准为年龄小于19岁且头部简明损伤量表评分为4、5或6。采用瓦哈卡-布林德分解法分析大量协变量对黑人和白人儿童死亡率差异的相对贡献。协变量被汇总为以下几类:“严重程度”“结构与过程”“机制”“人口统计学特征”和“保险”。符合分析条件的有7273名白人儿童和2320名黑人儿童。共有1661例死亡(17.3%)。白人儿童和黑人儿童的原始死亡率分别为15.6%和22.8%。归类为“严重程度”的因素占死亡率差异的95%,“机制”占13%,“保险”占5%,“人口统计学特征”占2%。“结构与过程”的贡献未达到统计学显著性。
损伤严重程度是造成黑人和白人儿童创伤性脑损伤死亡率差异的主要原因。机制、保险状况和性别所起的作用较小。由于保险状况与其他社会因素一样不能直接影响创伤存活率,其贡献的中介因素需要进一步研究。