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Early Days of Tenascin-R Research: Two Approaches Discovered and Shed Light on Tenascin-R.

作者信息

Rathjen Fritz G, Hodge Russell

机构信息

Department of Neuroscience, Max-Delbrück-Center for Molecular Medicine, Berlin, Germany.

出版信息

Front Immunol. 2021 Jan 8;11:612482. doi: 10.3389/fimmu.2020.612482. eCollection 2020.

DOI:10.3389/fimmu.2020.612482
PMID:33488619
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7820773/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/54cb/7820773/8b33fa6e834a/fimmu-11-612482-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/54cb/7820773/8b33fa6e834a/fimmu-11-612482-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/54cb/7820773/8b33fa6e834a/fimmu-11-612482-g001.jpg

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Chondroitin sulfate proteoglycan-5 forms perisynaptic matrix assemblies in the adult rat cortex.硫酸软骨素蛋白聚糖-5 在成年大鼠皮质中形成突触周基质组装。
Cell Signal. 2020 Oct;74:109710. doi: 10.1016/j.cellsig.2020.109710. Epub 2020 Jul 9.
2
Synaptic Specificity, Recognition Molecules, and Assembly of Neural Circuits.突触特异性、识别分子和神经回路的组装。
Cell. 2020 Apr 30;181(3):536-556. doi: 10.1016/j.cell.2020.04.008.
3
Loss of TNR causes a nonprogressive neurodevelopmental disorder with spasticity and transient opisthotonus.
Biomolecules. 2022 Nov 15;12(11):1689. doi: 10.3390/biom12111689.
4
Revisiting the Tenascins: Exploitable as Cancer Targets?重新审视腱糖蛋白：可用作癌症靶点吗？
Front Oncol. 2022 Jun 17;12:908247. doi: 10.3389/fonc.2022.908247. eCollection 2022.
5
The Extracellular Matrix Proteins Tenascin-C and Tenascin-R Retard Oligodendrocyte Precursor Maturation and Myelin Regeneration in a Cuprizone-Induced Long-Term Demyelination Animal Model.细胞外基质蛋白 tenascin-C 和 tenascin-R 延缓少突胶质前体细胞成熟和髓鞘再生在 cuprizone 诱导的长期脱髓鞘动物模型中。
Cells. 2022 May 28;11(11):1773. doi: 10.3390/cells11111773.
6
Tenascins Interfere With Remyelination in an Ex Vivo Cerebellar Explant Model of Demyelination.腱糖蛋白在脱髓鞘的体外小脑外植体模型中干扰髓鞘再生。
Front Cell Dev Biol. 2022 Mar 15;10:819967. doi: 10.3389/fcell.2022.819967. eCollection 2022.
7
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J Neurosci. 2022 Apr 6;42(14):3037-3048. doi: 10.1523/JNEUROSCI.1714-21.2022. Epub 2022 Feb 22.
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J Cell Sci. 2014 Feb 1;127(Pt 3):641-52. doi: 10.1242/jcs.137612. Epub 2013 Dec 11.
9
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10
Impaired presynaptic function and elimination of synapses at premature stages during postnatal development of the cerebellum in the absence of CALEB (CSPG5/neuroglycan C).在小脑发育的出生后早期阶段，由于缺乏 CALEB（CSPG5/神经糖蛋白 C），导致突触前功能受损和突触消除。
Eur J Neurosci. 2013 Nov;38(9):3270-80. doi: 10.1111/ejn.12313. Epub 2013 Jul 25.