Streitz J M, Streitz J M
Department of Surgery, New England Deaconess Hospital, Boston, Massachusetts.
J Urol. 1988 Apr;139(4):784-5. doi: 10.1016/s0022-5347(17)42636-x.
Polyuria of diabetes insipidus and psychogenic polydipsia can produce massive dilatation of the urinary tract in the absence of any mechanical obstruction. Renal failure in these cases is rare. We report the second case of nephrogenic diabetes insipidus with nonobstructive hydronephrotic renal damage. Temporary suprapubic drainage restored renal function to normal and decreased the upper tract dilatation. Renal function has been preserved for more than 10 years. Surgical intervention beyond temporary vesical drainage is unnecessary.
尿崩症和精神性多饮引起的多尿可在无任何机械性梗阻的情况下导致尿路广泛扩张。这些病例中肾衰竭罕见。我们报告第二例肾性尿崩症合并非梗阻性肾积水性肾损害。耻骨上临时引流使肾功能恢复正常并减轻了上尿路扩张。肾功能已维持超过10年。除临时膀胱引流外无需手术干预。