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The role of Hageman factor, prekallifrein, and high molecular weight kininogen in the generation of bradykinin and the initiation of coagulation and fibrinolysis.

作者信息

Kaplan A P, Meier H L, Mandle R J

出版信息

Monogr Allergy. 1977;12:120-30.

PMID:335210
Abstract
摘要

相似文献

1
The role of Hageman factor, prekallifrein, and high molecular weight kininogen in the generation of bradykinin and the initiation of coagulation and fibrinolysis.
Monogr Allergy. 1977;12:120-30.
2
Kininogen deficiency in Fitzgerald trait: role of high molecular weight kininogen in clotting and fibrinolysis.菲茨杰拉德性状中的激肽原缺乏:高分子量激肽原在凝血和纤维蛋白溶解中的作用。
J Lab Clin Med. 1976 Feb;87(2):327-37.
3
Initiation of the intrinsic coagulation and fibrinolytic pathways of man: the role of surfaces, hageman factor, prekallikrein, high molecular weight kininogen, and factor XI.人类内源性凝血和纤维蛋白溶解途径的启动:表面、哈格曼因子、前激肽释放酶、高分子量激肽原和因子XI的作用。
Prog Hemost Thromb. 1978;4:127-75.
4
Activation and function of human Hageman factor. The role of high molecular weight kininogen and prekallikrein.人凝血因子Ⅻ的激活与功能。高分子量激肽原和前激肽释放酶的作用。
J Clin Invest. 1977 Jul;60(1):18-31. doi: 10.1172/JCI108754.
5
Surface activation of blood coagulation, fibrinolysis and kinin formation.血液凝固、纤维蛋白溶解和激肽形成的表面激活。
Nature. 1980 Jul 31;286(5772):456-60. doi: 10.1038/286456a0.
6
Hageman factor-dependent pathways: mechanism of initiation and bradykinin formation.哈格曼因子依赖性途径:启动机制与缓激肽形成
Fed Proc. 1983 Nov;42(14):3123-7.
7
Prekallikrein activation by Hageman factor: potentiation by proteins in the absence of high molecular weight kininogen.哈格曼因子对前激肽释放酶的激活:在缺乏高分子量激肽原的情况下蛋白质的增强作用。
Trans Assoc Am Physicians. 1979;92:208-17.
8
Potentiation of the function of Hageman factor fragments by high molecular weight kininogen.高分子量激肽原对哈格曼因子片段功能的增强作用。
J Clin Invest. 1977 Jul;60(1):7-17. doi: 10.1172/JCI108770.
9
Fletcher factor deficiency. A diminished rate of Hageman factor activation caused by absence of prekallikrein with abnormalities of coagulation, fibrinolysis, chemotactic activity, and kinin generation.弗莱彻因子缺乏症。由于缺乏前激肽释放酶导致哈格曼因子激活速率降低,伴有凝血、纤维蛋白溶解、趋化活性和激肽生成异常。
J Clin Invest. 1974 Feb;53(2):622-33. doi: 10.1172/JCI107597.
10
Williams trait. Human kininogen deficiency with diminished levels of plasminogen proactivator and prekallikrein associated with abnormalities of the Hageman factor-dependent pathways.威廉姆斯特征。人类激肽原缺乏,纤溶酶原激活物和前激肽释放酶水平降低,与哈格曼因子依赖性途径异常相关。
J Clin Invest. 1975 Dec;56(6):1650-62. doi: 10.1172/JCI108247.

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Altered fibrin clot structure and dysregulated fibrinolysis contribute to thrombosis risk in severe COVID-19.纤维蛋白凝块结构改变和纤溶失调导致严重 COVID-19 血栓形成风险增加。
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The plasma kallikrein kinin system in severely ill and traumatised patients.重症及创伤患者的血浆激肽释放酶激肽系统
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