The role of Hageman factor, prekallifrein, and high molecular weight kininogen in the generation of bradykinin and the initiation of coagulation and fibrinolysis. - Suppr | 超能文献

The role of Hageman factor, prekallifrein, and high molecular weight kininogen in the generation of bradykinin and the initiation of coagulation and fibrinolysis.

作者信息

Kaplan A P, Meier H L, Mandle R J

出版信息

Monogr Allergy. 1977;12:120-30.

Abstract

摘要

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The role of Hageman factor, prekallifrein, and high molecular weight kininogen in the generation of bradykinin and the initiation of coagulation and fibrinolysis.

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Surface activation of blood coagulation, fibrinolysis and kinin formation.血液凝固、纤维蛋白溶解和激肽形成的表面激活。

Nature. 1980 Jul 31;286(5772):456-60. doi: 10.1038/286456a0.

6

Hageman factor-dependent pathways: mechanism of initiation and bradykinin formation.哈格曼因子依赖性途径：启动机制与缓激肽形成

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Prekallikrein activation by Hageman factor: potentiation by proteins in the absence of high molecular weight kininogen.哈格曼因子对前激肽释放酶的激活：在缺乏高分子量激肽原的情况下蛋白质的增强作用。

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Potentiation of the function of Hageman factor fragments by high molecular weight kininogen.高分子量激肽原对哈格曼因子片段功能的增强作用。

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Fletcher factor deficiency. A diminished rate of Hageman factor activation caused by absence of prekallikrein with abnormalities of coagulation, fibrinolysis, chemotactic activity, and kinin generation.弗莱彻因子缺乏症。由于缺乏前激肽释放酶导致哈格曼因子激活速率降低，伴有凝血、纤维蛋白溶解、趋化活性和激肽生成异常。

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Williams trait. Human kininogen deficiency with diminished levels of plasminogen proactivator and prekallikrein associated with abnormalities of the Hageman factor-dependent pathways.威廉姆斯特征。人类激肽原缺乏，纤溶酶原激活物和前激肽释放酶水平降低，与哈格曼因子依赖性途径异常相关。

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