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聚腺苷二磷酸核糖聚合酶抑制剂联合 EZH2 抑制剂在乳腺癌中的疗效取决于 BRCA 突变状态和微环境。

Efficacy of PARP inhibition combined with EZH2 inhibition depends on BRCA mutation status and microenvironment in breast cancer.

机构信息

Department of Molecular and Cellular Oncology, The University of Texas MD Anderson Cancer Center, Houston, TX, USA.

出版信息

FEBS J. 2021 May;288(9):2884-2887. doi: 10.1111/febs.15730. Epub 2021 Feb 11.

Abstract

The efficacy of the combination of a PARP inhibitor (PARPi) and an EZH2 inhibitor has been investigated in breast cancer cells with either BRCA1 mutation or BRCA2 mutation. However, earlier studies focused on the efficacy of this combination against BRCA-mutated but not BRCA-proficient breast cancer. Yang et al. observed that PARP1 depletion combined with EZH2 depletion via PRC2 depletion did not affect the growth of BRCA1/2 wild-type breast cancer cells in vitro. Moreover, Yang et al. reported that this combination stimulated synthetic viability of BRCA1/2-proficient breast cancer cells in vivo by regulating the tumor microenvironment to induce angiogenesis and differentiation of M2-type macrophages. The findings of Yang et al. provided evidence that both in vitro and animal models should be employed in the studies of PARPi combination therapies in order to involve the alteration of the tumor microenvironment in these investigations. These studies of PARP inhibition combined with EZH2 inhibition in breast cancer showed that this combination may benefit breast cancer patients carrying BRCA1-mutated tumor, but the combination may also enhance recurrence of BRCA2-mutated tumor and may even promote BRCA-proficient cancer cell survival. Therefore, BRCA1 mutation status should be used to select breast cancer patients for PARPi and EZH2 inhibitor combination treatment in clinical trials in the future.

摘要

在具有 BRCA1 突变或 BRCA2 突变的乳腺癌细胞中,已经研究了 PARP 抑制剂 (PARPi) 和 EZH2 抑制剂联合使用的疗效。然而,早期的研究侧重于该联合疗法对 BRCA 突变而不是 BRCA 有效的乳腺癌的疗效。Yang 等人观察到,通过 PRC2 耗竭,PARP1 耗竭与 EZH2 耗竭联合使用,不会影响体外 BRCA1/2 野生型乳腺癌细胞的生长。此外,Yang 等人报告称,这种组合通过调节肿瘤微环境来诱导血管生成和 M2 型巨噬细胞的分化,从而刺激 BRCA1/2 有效的乳腺癌细胞在体内的合成活力。Yang 等人的研究结果提供了证据,即应该在体外和动物模型中进行 PARPi 联合治疗的研究,以便在这些研究中涉及肿瘤微环境的改变。这些关于 PARP 抑制联合 EZH2 抑制在乳腺癌中的研究表明,这种联合治疗可能对携带 BRCA1 突变肿瘤的乳腺癌患者有益,但该联合治疗也可能增加 BRCA2 突变肿瘤的复发风险,甚至可能促进 BRCA 有效的癌细胞存活。因此,BRCA1 突变状态应在未来的临床试验中用于选择接受 PARPi 和 EZH2 抑制剂联合治疗的乳腺癌患者。

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