• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Correction to: Antibiotics-induced intestinal dysbacteriosis caused behavioral alternations and neuronal activation in different brain regions in mice.

作者信息

Wang Pan, Tu Ke, Cao Peng, Yang Yuefan, Zhang Hao, Qiu Xin-Tong, Zhang Ming-Ming, Wu Xiao-Jun, Yang Hui, Chen Tao

机构信息

Institute of Medical Research, Northwestern Polytechnical University, Xi`an, 710072, Shaanxi, People's Republic of China.

Department of Human Anatomy, Histology and Embryology & K.K. Leung Brain Research Centre, The Air Force Medical University, No. 169 Changle West Road, Xi'an, 710032, China.

出版信息

Mol Brain. 2021 Apr 13;14(1):69. doi: 10.1186/s13041-021-00781-y.

DOI:10.1186/s13041-021-00781-y
PMID:33849609
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8045266/
Abstract
摘要

相似文献

1
Correction to: Antibiotics-induced intestinal dysbacteriosis caused behavioral alternations and neuronal activation in different brain regions in mice.对《抗生素诱导的肠道菌群失调导致小鼠不同脑区的行为改变和神经元激活》一文的更正
Mol Brain. 2021 Apr 13;14(1):69. doi: 10.1186/s13041-021-00781-y.
2
Antibiotics-induced intestinal dysbacteriosis caused behavioral alternations and neuronal activation in different brain regions in mice.抗生素诱导的肠道菌群失调导致小鼠不同脑区的行为改变和神经元激活。
Mol Brain. 2021 Mar 6;14(1):49. doi: 10.1186/s13041-021-00759-w.
3
Effects of ceftriaxone-induced intestinal dysbacteriosis on dendritic cells of small intestine in mice.头孢曲松诱导的肠道菌群失调对小鼠小肠树突状细胞的影响。
Microbiol Immunol. 2013 Aug;57(8):561-8. doi: 10.1111/1348-0421.12068.
4
Intestinal dysbacteriosis mediates the reference memory deficit induced by anaesthesia/surgery in aged mice.肠道菌群失调介导麻醉/手术引起的老年小鼠参考记忆缺陷。
Brain Behav Immun. 2019 Aug;80:605-615. doi: 10.1016/j.bbi.2019.05.006. Epub 2019 May 4.
5
[The effect of enterococci on normalizing the intestinal microflora in experimental dysbacteriosis].
Zh Mikrobiol Epidemiol Immunobiol. 1989 Sep(9):3-6.
6
Intestinal dysbacteriosis activates tumor-associated macrophages to promote epithelial-mesenchymal transition of colorectal cancer.肠道菌群失调激活肿瘤相关巨噬细胞促进结直肠癌细胞上皮间质转化。
Innate Immun. 2018 Nov;24(8):480-489. doi: 10.1177/1753425918801496. Epub 2018 Sep 23.
7
Effects of ceftriaxone induced intestinal dysbacteriosis on lymphocytes in different tissues in mice.头孢曲松诱导的肠道菌群失调对小鼠不同组织中淋巴细胞的影响。
Immunobiology. 2016 Sep;221(9):994-1000. doi: 10.1016/j.imbio.2016.04.003. Epub 2016 May 7.
8
[Intestinal dysbacteriosis promotes intestinal intraepithelial T lymphocyte activation and proinflammatory cytokine secretion in mice].[肠道菌群失调促进小鼠肠道上皮内T淋巴细胞活化及促炎细胞因子分泌]
Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2016 Aug;32(8):1031-5.
9
Intestinal dysbacteriosis contributes to decreased intestinal mucosal barrier function and increased bacterial translocation.肠道菌群失调可导致肠道黏膜屏障功能下降和细菌易位增加。
Lett Appl Microbiol. 2014 Apr;58(4):384-92. doi: 10.1111/lam.12201. Epub 2013 Dec 19.
10
[Influence of intestinal dysbacteriosis on immune and hematopoietec function in mice].[肠道菌群失调对小鼠免疫和造血功能的影响]
Zhonghua Er Ke Za Zhi. 2004 Sep;42(9):708-11.

引用本文的文献

1
Antibiotic-induced gut dysbiosis and cognitive, emotional, and behavioral changes in rodents: a systematic review and meta-analysis.抗生素诱导的啮齿动物肠道微生物群失调与认知、情绪及行为变化：一项系统评价和荟萃分析
Front Neurosci. 2023 Sep 1;17:1237177. doi: 10.3389/fnins.2023.1237177. eCollection 2023.
2
Enterochromaffin Cells: Sentinels to Gut Microbiota in Hyperalgesia?肠嗜铬细胞：在痛觉过敏中充当肠道微生物群的哨兵？
Front Cell Infect Microbiol. 2021 Oct 14;11:760076. doi: 10.3389/fcimb.2021.760076. eCollection 2021.

本文引用的文献

1
Antibiotics-induced intestinal dysbacteriosis caused behavioral alternations and neuronal activation in different brain regions in mice.抗生素诱导的肠道菌群失调导致小鼠不同脑区的行为改变和神经元激活。
Mol Brain. 2021 Mar 6;14(1):49. doi: 10.1186/s13041-021-00759-w.