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1
Loss of Transforming Growth Factor Beta Signaling in Aortic Smooth Muscle Cells Causes Endothelial Dysfunction and Aortic Hypercontractility.
Arterioscler Thromb Vasc Biol. 2021 Jun;41(6):1956-1971. doi: 10.1161/ATVBAHA.121.315878. Epub 2021 Apr 15.
2
Proteomics reveals Rictor as a noncanonical TGF-β signaling target during aneurysm progression in Marfan mice.
Am J Physiol Heart Circ Physiol. 2018 Nov 1;315(5):H1112-H1126. doi: 10.1152/ajpheart.00089.2018. Epub 2018 Jul 13.
3
TGF-β (Transforming Growth Factor-β) Signaling Protects the Thoracic and Abdominal Aorta From Angiotensin II-Induced Pathology by Distinct Mechanisms.
Arterioscler Thromb Vasc Biol. 2017 Nov;37(11):2102-2113. doi: 10.1161/ATVBAHA.117.309401. Epub 2017 Jul 20.
4
Smad4 Deficiency in Smooth Muscle Cells Initiates the Formation of Aortic Aneurysm.
Circ Res. 2016 Feb 5;118(3):388-99. doi: 10.1161/CIRCRESAHA.115.308040. Epub 2015 Dec 23.
5
Vascular smooth muscle cell phenotypic changes in patients with Marfan syndrome.
Arterioscler Thromb Vasc Biol. 2015 Apr;35(4):960-72. doi: 10.1161/ATVBAHA.114.304412. Epub 2015 Jan 15.
6
Role of Macrophage Socs3 in the Pathogenesis of Aortic Dissection.
J Am Heart Assoc. 2018 Jan 17;7(2):e007389. doi: 10.1161/JAHA.117.007389.
8
Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice.
Circ Heart Fail. 2015 Jul;8(4):788-98. doi: 10.1161/CIRCHEARTFAILURE.114.001963. Epub 2015 May 18.

引用本文的文献

1
Endothelial Cell Senescence in Marfan Syndrome: Pathogenesis and Therapeutic Potential of TGF-β Pathway Inhibition.
J Am Heart Assoc. 2025 May 6;14(9):e037826. doi: 10.1161/JAHA.124.037826. Epub 2025 Apr 16.
2
Chemokine (C-C Motif) Ligand 2 Expressing Adventitial Fibroblast Expansion During Loeys-Dietz Syndrome Aortic Aneurysm Formation.
Arterioscler Thromb Vasc Biol. 2025 May;45(5):722-742. doi: 10.1161/ATVBAHA.124.322069. Epub 2025 Mar 20.
5
Understanding genomic medicine for thoracic aortic disease through the lens of induced pluripotent stem cells.
Front Cardiovasc Med. 2024 Feb 19;11:1349548. doi: 10.3389/fcvm.2024.1349548. eCollection 2024.
6
7
Smooth Muscle Heterogeneity and Plasticity in Health and Aortic Aneurysmal Disease.
Int J Mol Sci. 2023 Jul 20;24(14):11701. doi: 10.3390/ijms241411701.
8
Molecular Mechanisms in Genetic Aortopathy-Signaling Pathways and Potential Interventions.
Int J Mol Sci. 2023 Jan 16;24(2):1795. doi: 10.3390/ijms24021795.
9
The emerging role of endothelial cells in the pathogenesis of thoracic aortic aneurysm and dissection.
Eur Heart J. 2023 Apr 7;44(14):1262-1264. doi: 10.1093/eurheartj/ehac771.
10
Endothelial dysfunction in Marfan syndrome mice is restored by resveratrol.
Sci Rep. 2022 Dec 28;12(1):22504. doi: 10.1038/s41598-022-26662-5.

本文引用的文献

1
2
Promoters to Study Vascular Smooth Muscle.
Arterioscler Thromb Vasc Biol. 2019 Apr;39(4):603-612. doi: 10.1161/ATVBAHA.119.312449.
3
Endothelial to Mesenchymal Transition in Cardiovascular Disease: JACC State-of-the-Art Review.
J Am Coll Cardiol. 2019 Jan 22;73(2):190-209. doi: 10.1016/j.jacc.2018.09.089.
4
LMO7 Is a Negative Feedback Regulator of Transforming Growth Factor β Signaling and Fibrosis.
Circulation. 2019 Jan 29;139(5):679-693. doi: 10.1161/CIRCULATIONAHA.118.034615.
5
Endothelial-Vascular Smooth Muscle Cells Interactions in Atherosclerosis.
Front Cardiovasc Med. 2018 Oct 23;5:151. doi: 10.3389/fcvm.2018.00151. eCollection 2018.
6
The dominant protein phosphatase PP1c isoform in smooth muscle cells, PP1cβ, is essential for smooth muscle contraction.
J Biol Chem. 2018 Oct 26;293(43):16677-16686. doi: 10.1074/jbc.RA118.003083. Epub 2018 Sep 5.
7
Endothelial HIF-2α contributes to severe pulmonary hypertension due to endothelial-to-mesenchymal transition.
Am J Physiol Lung Cell Mol Physiol. 2018 Feb 1;314(2):L256-L275. doi: 10.1152/ajplung.00096.2017. Epub 2017 Oct 26.
8
Endothelial-to-mesenchymal transition in cardiovascular diseases: Developmental signaling pathways gone awry.
Dev Dyn. 2018 Mar;247(3):492-508. doi: 10.1002/dvdy.24589. Epub 2017 Oct 25.
9
Deficient Circumferential Growth Is the Primary Determinant of Aortic Obstruction Attributable to Partial Elastin Deficiency.
Arterioscler Thromb Vasc Biol. 2017 May;37(5):930-941. doi: 10.1161/ATVBAHA.117.309079. Epub 2017 Mar 2.

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