Bergström J, Persson L, Preber H
Department of Periodontology, School of Dentistry, Karolinska Institutet, Stockholm, Sweden.
Scand J Dent Res. 1988 Feb;96(1):34-9. doi: 10.1111/j.1600-0722.1988.tb01405.x.
The influence of cigarette smoking on the vascular reaction during plaque induced gingivitis was studied in humans for 28 days. Sixteen healthy dental students, 8 smokers and 8 non-smokers, aged 19-42 yr, volunteered for the experiment. A numerical method was used for the evaluation of the vascular reaction. With the aid of stereophotographs changes in the number of gingival vessels were followed during the experiment. It was found that the number of vessels identified increased over time during the experiment in both smokers and non-smokers. However, in spite of the fact that the plaque accumulation rate was equal, the vascular reaction was less pronounced in smokers. At the end of the experiment after 28 days the intensity of the vascular reaction in smokers was only 50% of that observed in non-smokers. The difference was statistically significant at the P-level of 0.05. One week after termination of the experiment and reinstitution of oral hygiene the number of gingival vessels equaled the pre-experimental values in both groups. The results indicate that the vascular reaction associated with plaque induced gingivitis is suppressed in smokers.
在人体中研究了吸烟对菌斑性牙龈炎期间血管反应的影响，为期28天。16名年龄在19至42岁之间的健康牙科学生，8名吸烟者和8名不吸烟者，自愿参与了该实验。采用数值方法评估血管反应。借助立体照片，在实验过程中跟踪牙龈血管数量的变化。结果发现，在实验过程中，吸烟者和不吸烟者识别出的血管数量均随时间增加。然而，尽管菌斑积累率相同，但吸烟者的血管反应不太明显。实验28天后结束时，吸烟者血管反应的强度仅为不吸烟者的50%。在P值为0.05时，差异具有统计学意义。实验终止并重新建立口腔卫生一周后，两组牙龈血管数量均恢复到实验前的值。结果表明，吸烟者中与菌斑性牙龈炎相关的血管反应受到抑制。
