Debnath Ujjwal K
Professor of Orthopaedics, Jagannath Gupta Institute of Medical Sciences, Kolkata.
Consultant Orthopaedic & Spine, Surgeon, Fortis Hospital, Kolkata.
J Clin Orthop Trauma. 2021 Jul 30;21:101535. doi: 10.1016/j.jcot.2021.101535. eCollection 2021 Oct.
Lumbar pars interarticularis (PI) injury or spondylolysis occurs only in humans. This represents a stress fracture of the PI. Excessive loading in repetitive hyperextension is a significant risk factor and occurs most commonly at L5 followed by L4. It is bilateral in 80% of symptomatic cases but can be unilateral defect as well which runs a more benign course. Symptoms of low back pain relating to this lesion are more common in young athletes involved in trunk twisting sports. Like other stress fractures, the pain may come on abruptly or more insidiously over time and only related to certain activities. The pathologic progression starts with a stress reaction in the pars, progressing to an incomplete stress fracture, and then a complete pars fracture. Diagnosis is dependent on clinical examination and radiological imaging studies (plain radiography, computed tomography (CT) scans and magnetic resonance imaging (MRI) scans). Treatment is dependent on symptoms as well as radiographic stage of the lesion. Conservative management is the mainstay of treating early lesions. A comprehensive rehabilitation program incorporates core spinal stabilization exercises. Athletes should not return to sports until pain free. Professional sporting individuals are at increased risk of failure of resolution of symptoms that may require early surgical repair of the PI defect. Modified Buck's technique & pedicle screw-hook constructs for direct repair has a high success rate in patients who have persistent low back pain. Minimally invasive lumbar pars defect repair has given similar successful outcome with added advantage of minimizing muscle injury, preserving the adjacent joint and reduced hospital stay. Functional outcome is evaluated using the Visual Analogue Scale (VAS) for back pain, Oswestry Disability Index (ODI) and 36-Item Short-Form Health Survey (SF-36). Preoperative ODI and SF-36 physical component scores (PCS) are significant predictor of a good functional outcome.
腰椎峡部裂(PI)损伤或椎弓根崩裂仅发生于人类。这是一种峡部的应力性骨折。反复过度伸展时的过度负荷是一个重要危险因素,最常发生于L5,其次是L4。80%有症状的病例为双侧病变,但也可为单侧缺损,其病程相对良性。与该病变相关的下腰痛症状在从事躯干扭转运动的年轻运动员中更为常见。与其他应力性骨折一样,疼痛可能突然出现,也可能随着时间的推移更隐匿地出现,且仅与某些活动有关。病理进展始于峡部的应力反应,进展为不完全应力性骨折,然后发展为完全峡部骨折。诊断依赖于临床检查和影像学研究(X线平片、计算机断层扫描(CT)和磁共振成像(MRI)扫描)。治疗取决于症状以及病变的影像学分期。保守治疗是早期病变治疗的主要方法。一个全面的康复计划包括核心脊柱稳定练习。运动员在无痛之前不应恢复运动。职业运动员症状缓解失败的风险增加,可能需要早期手术修复PI缺损。改良的巴克技术和椎弓根螺钉钩结构用于直接修复,对于持续存在下腰痛的患者成功率较高。微创腰椎峡部缺损修复取得了类似的成功结果,还具有减少肌肉损伤、保留相邻关节和缩短住院时间的额外优势。使用视觉模拟量表(VAS)评估背痛、Oswestry功能障碍指数(ODI)和36项简短健康调查(SF-36)来评估功能结果。术前ODI和SF-36身体成分评分(PCS)是良好功能结果的重要预测指标。