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阿育吠陀疗法治疗肢带型肌营养不良——病例报告

Ayurvedic management in limb girdle muscular dystrophy - A case report.

作者信息

Rajoria Kshipra, Singh Sarvesh Kumar, Dadhich Suman

机构信息

Department of Panchakarma, National Institute of Ayurveda, Jaipur, India.

Department of Panchakarma, National Institute of Ayurveda, Jaipur, India.

出版信息

J Ayurveda Integr Med. 2022 Jan-Mar;13(1):100486. doi: 10.1016/j.jaim.2021.07.002. Epub 2021 Dec 24.

DOI:10.1016/j.jaim.2021.07.002
PMID:34961685
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8728082/
Abstract

Limb girdle muscular dystrophy (LGMD) is a type of Muscular dystrophy (MD), heterogeneous devastating complex genetic disorders causing progressive weakness and degeneration of muscles. LGMD is hereditary autosomal diseases characterized by weak and wasteful limb girdle muscles. The available management of LGMD in biomedicine is unsatisfactory. Here we present a case of LGMD managed with combinations of Ayurvedic oral medicines and Panchakarma procedures. The Ayurvedic diagnosis of the condition was considered as Mansagata Vata (∼neuromuscular diseases), a type of Vatavyadhi (∼neuromusculo skeleton disorders). The patient was treated with Shalishashtika Pinda Swedana and Mustadi Yapana Basti for the duration of 16 days along with following Ayurvedic oral medicines: Yograj Guggulu 500 mg with 40 ml Dashamoola Kwatha, Ekangaveera Rasa 125 mg with honey, a combination of Ashwagandha Churna -2g, Satavari Churna - 2g, and Sankha Bhasma 500 mg with milk, Narsinha Churna- 3g and Ashwagandhavleha- 5g with milk. All medicines were given twice a day. Patient's condition was assessed for symptoms of pain, walking distance, power and reflexes of both upper and lower limb and psedohypertrophy of both calf muscles. Serum Creatine Phoshphokinase (S.CPK) level and electromyography (EMG) were also measured. There was symptomatic improvement in the patient's condition and reduction in S.CPK level. The study suggests that LGMD can be satisfactorily managed with Ayurvedic oral medicines and Panchakarma therapy.

摘要

肢带型肌营养不良(LGMD)是一种肌营养不良(MD),是一组异质性的、具有破坏性的复杂遗传性疾病,可导致肌肉进行性无力和退化。LGMD是遗传性常染色体疾病,其特征是肢带肌肉无力和萎缩。目前生物医学对LGMD的治疗并不令人满意。在此,我们报告一例采用阿育吠陀口服药物和五种疗法相结合治疗的LGMD病例。该疾病的阿育吠陀诊断被认为是曼萨加塔瓦塔(类似于神经肌肉疾病),一种瓦塔维迪(类似于神经肌肉骨骼疾病)。患者接受了16天的沙利沙什蒂卡药饼熏蒸和穆斯塔迪药油灌肠治疗,同时服用以下阿育吠陀口服药物:500毫克瑜伽王古古卢与40毫升十味药汤、125毫克埃康加维拉拉萨与蜂蜜、2克阿育吠陀根粉、2克沙塔瓦里粉和500毫克贝壳灰与牛奶、3克那罗辛哈粉和5克阿育吠陀膏与牛奶。所有药物均每日服用两次。对患者的病情进行了评估,包括疼痛症状、行走距离、上下肢的力量和反射以及双侧小腿肌肉的假性肥大。还测量了血清肌酸磷酸激酶(S.CPK)水平和肌电图(EMG)。患者的病情有症状改善,S.CPK水平降低。该研究表明,LGMD可以通过阿育吠陀口服药物和五种疗法得到满意的治疗。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7e93/8728082/0a7d5500795e/figs8.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7e93/8728082/d9a0658094ac/ga1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7e93/8728082/2c8baf6e1e06/figs1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7e93/8728082/255abdb824a6/figs2.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7e93/8728082/0a7d5500795e/figs8.jpg

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