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Clin Exp Immunol. 1987 Oct;70(1):217-21.
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引用本文的文献

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The interleukins in acquired disease.后天性疾病中的白细胞介素。
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本文引用的文献

1
Immunogenic cells in the regional lymph nodes after painting with the contact sensitizers picryl chloride and oxazolone: evidence for the presence of IgM antibody on their surface.在用接触性致敏剂氯化苦基和恶唑酮涂抹后区域淋巴结中的免疫原性细胞:其表面存在IgM抗体的证据。
Immunology. 1983 Mar;48(3):561-9.
2
Suppressor cells and the handling of antigen.抑制细胞与抗原处理
Immunol Rev. 1980;50:3-45. doi: 10.1111/j.1600-065x.1980.tb00306.x.
3
The activity of natural cytotoxic cells is augmented by interleukin 2 and interleukin 3.白细胞介素2和白细胞介素3可增强自然杀伤细胞的活性。
J Exp Med. 1983 Mar 1;157(3):1070-5. doi: 10.1084/jem.157.3.1070.
4
T helper factor in contact sensitivity: antigen-specific I-A+ helper factor is made by an Lyt-1+2-, I-A+, I-J- T cell.接触敏感性中的辅助性T细胞因子:抗原特异性I-A+辅助因子由Lyt-1+2-、I-A+、I-J- T细胞产生。
Immunology. 1984 Jun;52(2):261-7.
5
Topical immunotherapy of basal cell carcinomas with dinitrochlorobenzene.用二硝基氯苯对基底细胞癌进行局部免疫治疗。
Cancer Res. 1973 Nov;33(11):3036-42.
6
Contact sensitivity in the mouse. X. Non-specific cytotoxicity of T blasts in the draining lymph nodes.小鼠的接触敏感性。X. 引流淋巴结中T母细胞的非特异性细胞毒性。
Immunology. 1973 Sep;25(3):471-83.
7
Acquired immunological tolerance of foreign cells is impaired by recombinant interleukin 2 or vitamin A acetate.重组白细胞介素2或醋酸维生素A会损害对外源细胞的获得性免疫耐受性。
Proc Natl Acad Sci U S A. 1985 Jan;82(2):536-8. doi: 10.1073/pnas.82.2.536.
8
T-cell depletion of allogeneic bone marrow prevents acceleration of graft-versus-host disease induced by exogenous interleukin 2.异体骨髓的T细胞清除可防止外源性白细胞介素2诱导的移植物抗宿主病加速。
Cell Immunol. 1986 Dec;103(2):476-80. doi: 10.1016/0008-8749(86)90108-5.
9
Acute cutaneous graft-versus-host disease to minor histocompatibility antigens in a murine model. Evidence that large granular lymphocytes are effector cells in the immune response.小鼠模型中针对次要组织相容性抗原的急性皮肤移植物抗宿主病。大颗粒淋巴细胞是免疫反应中效应细胞的证据。
Lab Invest. 1986 Jul;55(1):35-42.
10
Antitumor efficacy of lymphokine-activated killer cells and recombinant interleukin-2 in vivo: survival benefit and mechanisms of tumor escape in mice undergoing immunotherapy.淋巴因子激活的杀伤细胞和重组白细胞介素-2在体内的抗肿瘤疗效:接受免疫治疗小鼠的生存获益及肿瘤逃逸机制
Cancer Res. 1986 Feb;46(2):676-83.

在用接触致敏剂免疫后,小鼠局部淋巴结中淋巴因子激活的杀伤细胞(LAK)活性增强。

Increased lymphokine activated killer (LAK) activity in the regional lymph nodes of mice following immunization with contact sensitizing agents.

作者信息

Gao L Q, Asherson G L, Malkovsky M

机构信息

Division of Immunological Medicine, Clinical Research Centre, Harrow, UK.

出版信息

Clin Exp Immunol. 1987 Oct;70(1):217-21.

PMID:3500814
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1542224/
Abstract

The regional lymph nodes of mice show increased IL-2 activated killer (LAK) activity following immunization with the contact sensitizing agents, 'oxazolone' and picryl chloride. This is best elicited with 500 mu/ml human recombinant IL-2 and can be demonstrated with both YAC-1 and K562 targets. There is also a lesser increase in NK activity.

摘要

在用接触致敏剂“恶唑酮”和苦味酸氯化物免疫后,小鼠的局部淋巴结显示出白细胞介素-2激活的杀伤细胞(LAK)活性增加。这在用500微克/毫升人重组白细胞介素-2时最易引发,并且用YAC-1和K562靶细胞均可证实。自然杀伤细胞(NK)活性也有较小程度的增加。