Medina Giovanna, Bartolozzi Arthur R, Spencer Jacob A, Morgan Craig
Jefferson Health 3B Orthopaedics, Philadelphia, Pennsylvania.
STAR Orthopedics, Palm Desert, California.
JBJS Rev. 2022 Mar 18;10(3):01874474-202203000-00014. doi: e21.00194.
»: The thrower's shoulder has been a subject of great interest for many decades. Different theories have been proposed to clarify the pathophysiology, clinical presentation, and treatment options for this condition. In this review article, we summarize the relevant anatomy and pathophysiology and how these translate into signs, symptoms, and imaging findings. Also, a historical review of the treatment methodologies in the setting of an evolving concept is presented.
»: The initial event in the cascade is thickening and contracture of the posteroinferior capsule resulting from repetitive tensile forces during the deceleration phase of throwing. This is known as "the essential lesion" and is clinically perceived as glenohumeral internal rotation deficit (GIRD), and a Bennett lesion may be found on radiographs.
»: Change in the glenohumeral contact point leads to a series of adaptations that are beneficial for the mechanics of throwing, specifically in achieving the so-called "slot," which will maximize throwing performance.
»: The complexity of the throwing shoulder is the result of an interplay of the different elements described in the cascade, as well as other factors such as pectoralis minor tightness and scapular dyskinesis. However, it is still unclear which event is the tipping point that breaks the balance between these adaptations and triggers the shift from an asymptomatic shoulder to a painful disabled joint that can jeopardize the career of a throwing athlete. Consequences are rotator cuff impingement and tear, labral injury, and scapular dyskinesis, which are seen both clinically and radiographically.
»: A thorough understanding of the pathologic cascade is paramount for professionals who care for throwing athletes. The successful treatment of this condition depends on correct identification of the point in the cascade that is disturbed. The typical injuries described in the throwing shoulder rarely occur in isolation; thus, an overlap of symptoms and clinical findings is common.
»: The rationale for treatment is based on the pathophysiologic biomechanics and should involve stretching, scapular stabilization, and core and lower-body strengthening, as well as correction of throwing mechanics, integrating the entire kinetic chain. When nonoperative treatment is unsuccessful, surgical options should be tailored for the specific changes within the pathologic cascade that are causing a dysfunctional throwing shoulder.
几十年来,投掷运动员的肩部一直备受关注。人们提出了不同的理论来阐明这种情况的病理生理学、临床表现和治疗方案。在这篇综述文章中,我们总结了相关的解剖结构和病理生理学,以及它们如何转化为体征、症状和影像学表现。此外,还对在不断演变的概念背景下的治疗方法进行了历史回顾。
该病理过程的初始事件是在投掷减速阶段由于重复性拉力导致后下关节囊增厚和挛缩。这被称为“基本病变”,临床上表现为肩肱关节内旋不足(GIRD),X线片上可能会发现贝内特损伤。
肩肱关节接触点的改变会引发一系列有利于投掷力学的适应性变化,特别是在实现所谓的“投球槽”方面,这将使投掷表现最大化。
投掷肩的复杂性是由该病理过程中描述的不同因素相互作用的结果,以及其他因素,如胸小肌紧张和肩胛骨运动障碍。然而,仍不清楚哪个事件是打破这些适应性变化之间平衡并触发从无症状肩部向可能危及投掷运动员职业生涯的疼痛残疾关节转变的临界点。其后果是肩袖撞击和撕裂、盂唇损伤以及肩胛骨运动障碍,这些在临床和影像学上都可见。
对于照顾投掷运动员的专业人员来说,全面了解病理过程至关重要。这种情况的成功治疗取决于正确识别病理过程中受干扰的点。投掷肩中描述的典型损伤很少单独发生;因此,症状和临床发现重叠很常见。
治疗的基本原理基于病理生理生物力学,应包括拉伸、肩胛骨稳定、核心和下半身强化,以及纠正投掷力学,整合整个动力链。当非手术治疗不成功时,手术选择应针对导致投掷肩功能障碍的病理过程中的特定变化进行调整。