Variend S, Forrest A R
Arch Dis Child. 1987 Apr;62(4):417-8. doi: 10.1136/adc.62.4.417.
Carboxyhaemoglobin measured in 50 infant deaths showed no significant difference between home and hospital deaths nor between explained and unexplained deaths. Carbon monoxide toxicity is unlikely to have an important role in the pathogenesis of sudden infant deaths. The generally low carboxyhaemoglobin concentrations are probably due to endogenous production.
对50例婴儿死亡病例所测的碳氧血红蛋白显示,家中死亡与医院死亡之间、死因明确与死因不明的死亡之间均无显著差异。一氧化碳中毒在婴儿猝死的发病机制中不太可能起重要作用。碳氧血红蛋白浓度普遍较低可能是内源性产生所致。