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转录组谱分析突出了克里米亚-刚果出血热病毒感染后调节的生物学过程和 III 型干扰素抗病毒反应。

Transcriptome profiling highlights regulated biological processes and type III interferon antiviral responses upon Crimean-Congo hemorrhagic fever virus infection.

机构信息

State Key Laboratory of Virology and National Virus Resource Center, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071/430207, China; University of Chinese Academy of Sciences, Beijing, 101408, China.

State Key Laboratory of Virology and National Virus Resource Center, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071/430207, China.

出版信息

Virol Sin. 2023 Feb;38(1):34-46. doi: 10.1016/j.virs.2022.09.002. Epub 2022 Sep 6.

DOI:10.1016/j.virs.2022.09.002
PMID:36075566
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10006212/
Abstract

Crimean-Congo hemorrhagic fever virus (CCHFV) is a biosafety level-4 (BSL-4) pathogen that causes Crimean-Congo hemorrhagic fever (CCHF) characterized by hemorrhagic manifestation, multiple organ failure and high mortality rate, posing great threat to public health. Despite the recently increasing research efforts on CCHFV, host cell responses associated with CCHFV infection remain to be further characterized. Here, to better understand the cellular response to CCHFV infection, we performed a transcriptomic analysis in human kidney HEK293 ​cells by high-throughput RNA sequencing (RNA-seq) technology. In total, 496 differentially expressed genes (DEGs), including 361 up-regulated and 135 down-regulated genes, were identified in CCHFV-infected cells. These regulated genes were mainly involved in host processes including defense response to virus, response to stress, regulation of viral process, immune response, metabolism, stimulus, apoptosis and protein catabolic process. Therein, a significant up-regulation of type III interferon (IFN) signaling pathway as well as endoplasmic reticulum (ER) stress response was especially remarkable. Subsequently, representative DEGs from these processes were well validated by RT-qPCR, confirming the RNA-seq results and the typical regulation of IFN responses and ER stress by CCHFV. Furthermore, we demonstrate that not only type I but also type III IFNs (even at low dosages) have substantial anti-CCHFV activities. Collectively, the data may provide new and comprehensive insights into the virus-host interactions and particularly highlights the potential role of type III IFNs in restricting CCHFV, which may help inform further mechanistic delineation of the viral infection and development of anti-CCHFV strategies.

摘要

克里米亚-刚果出血热病毒(CCHFV)是一种生物安全 4 级(BSL-4)病原体,可引起克里米亚-刚果出血热(CCHF),其特征为出血表现、多器官衰竭和高死亡率,对公共卫生构成巨大威胁。尽管最近对 CCHFV 的研究有所增加,但与 CCHFV 感染相关的宿主细胞反应仍需进一步表征。在这里,为了更好地了解细胞对 CCHFV 感染的反应,我们通过高通量 RNA 测序(RNA-seq)技术对人肾 HEK293 细胞进行了转录组分析。总共鉴定出 496 个差异表达基因(DEGs),包括 361 个上调基因和 135 个下调基因,这些基因在 CCHFV 感染细胞中受到调控。这些调节基因主要参与宿主过程,包括对病毒的防御反应、应激反应、病毒过程的调节、免疫反应、代谢、刺激、凋亡和蛋白质分解过程。其中,III 型干扰素(IFN)信号通路和内质网(ER)应激反应的显著上调尤为显著。随后,通过 RT-qPCR 对这些过程中的代表性 DEGs 进行了验证,证实了 RNA-seq 结果以及 CCHFV 对 IFN 反应和 ER 应激的典型调节。此外,我们证明了不仅是 I 型干扰素,而且 III 型干扰素(即使在低剂量下)也具有显著的抗 CCHFV 活性。总之,这些数据可能为病毒-宿主相互作用提供新的和全面的见解,特别是强调了 III 型干扰素在限制 CCHFV 方面的潜在作用,这可能有助于进一步阐明病毒感染的机制和开发抗 CCHFV 策略。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/0359ccd3bf83/gr8.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/f69b2ba6cf65/gr5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/47299dd774d7/gr6.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/bab9c900e3cc/gr7.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/0359ccd3bf83/gr8.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/789e76d34887/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/2597c37574f5/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/d4870301ca1d/gr3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/fe61ab0c577a/gr4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/f69b2ba6cf65/gr5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/47299dd774d7/gr6.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/bab9c900e3cc/gr7.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9c77/10006212/0359ccd3bf83/gr8.jpg

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