Gardaz J P, Forster A, Suter P M
Can Anaesth Soc J. 1979 Jan;26(1):34-7. doi: 10.1007/BF03039451.
We have presented a case of acute fulminating pulmonary oedema, not cardiogenic in origin, occurring at the end of anaesthesia. This precise diagnosis was made with the help of measurement of the capillary wedge pressure and cardiac output as well as analysis of the alevolar oedema fluid. The haemodynamic values and the gas exchange, as measured under various ventilatory patterns, confirms the usefulness of continuous positive pressure ventilation (CPPV). In the genesis of acute pulmonary oedema as described, the role of hypoxia and acute obstruction of the upper airways as presented by the patient is discussed in the light of the present proposed theories.
我们报告了一例急性暴发性肺水肿病例,其并非心源性,发生在麻醉末期。借助测量毛细血管楔压和心输出量以及分析肺泡水肿液做出了这一精确诊断。在各种通气模式下测得的血流动力学值和气体交换证实了持续正压通气(CPPV)的有效性。根据目前提出的理论,讨论了在所述急性肺水肿的发生过程中,患者所表现出的缺氧和上呼吸道急性梗阻的作用。