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1
Increases in dietary phosphate levels can augment atrial arrhythmias.饮食中磷酸盐水平的升高会加剧房性心律失常。
Acta Physiol (Oxf). 2023 Jun;238(2):e13978. doi: 10.1111/apha.13978. Epub 2023 Apr 30.
2
High-phosphate diet causes atrial remodeling and increases atrial fibrillation vulnerability via STAT3/NF-κB signaling and oxidative stress.高磷饮食通过 STAT3/NF-κB 信号通路和氧化应激引起心房重构,增加心房颤动易感性。
Acta Physiol (Oxf). 2023 Jun;238(2):e13964. doi: 10.1111/apha.13964. Epub 2023 Mar 31.
3
Arrhythmias Originating in the Atria.源于心房的心律失常。
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4
The atria: from morphology to function.心房:从形态学到功能。
J Cardiovasc Electrophysiol. 2011 Feb;22(2):223-35. doi: 10.1111/j.1540-8167.2010.01887.x. Epub 2010 Aug 31.
5
ATRIAL HEART SOUNDS IN ATRIAL FIBRILLATION AND FLUTTER.心房颤动和心房扑动时的心房心音。
Circulation. 1964 Dec;30:893-6. doi: 10.1161/01.cir.30.6.893.
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Vitamin D Levels in Patients with Atrial Fibrillation.心房颤动患者的维生素D水平
Ann Noninvasive Electrocardiol. 2016 Nov;21(6):622-623. doi: 10.1111/anec.12306. Epub 2016 Jun 20.
7
Arrhythmias in Patients with Atrial Defects.房间隔缺损患者的心律失常
Card Electrophysiol Clin. 2017 Jun;9(2):235-244. doi: 10.1016/j.ccep.2017.02.006.
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Atrial Arrhythmias in Patients With an ASD: Where to From Here?房间隔缺损患者的房性心律失常:何去何从?
JACC Clin Electrophysiol. 2020 May;6(5):549-551. doi: 10.1016/j.jacep.2020.01.012.
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Mechano-electric feedback and atrial fibrillation.机械电反馈与心房颤动
Prog Biophys Mol Biol. 2003 May-Jul;82(1-3):137-49. doi: 10.1016/s0079-6107(03)00011-7.
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Impact of Initial Extensive Ablation on Left Atrial Gaps During Redo Procedures and Subsequent Outcomes in Persistent Atrial Fibrillation.初次广泛消融对持续性心房颤动再次手术时左心房间隙及后续结局的影响
Circ Arrhythm Electrophysiol. 2025 Jun;18(6):e013612. doi: 10.1161/CIRCEP.124.013612. Epub 2025 Jun 2.

本文引用的文献

1
Quantifying a spectrum of clinical response in atrial tachyarrhythmias using spatiotemporal synchronization of electrograms.使用电描记图的时空同步量化房性心动过速的临床反应谱。
Europace. 2023 May 19;25(5). doi: 10.1093/europace/euad055.
2
High-phosphate diet causes atrial remodeling and increases atrial fibrillation vulnerability via STAT3/NF-κB signaling and oxidative stress.高磷饮食通过 STAT3/NF-κB 信号通路和氧化应激引起心房重构,增加心房颤动易感性。
Acta Physiol (Oxf). 2023 Jun;238(2):e13964. doi: 10.1111/apha.13964. Epub 2023 Mar 31.
3
Kidney glycolysis serves as a mammalian phosphate sensor that maintains phosphate homeostasis.肾脏糖酵解作为哺乳动物的磷酸盐感受器,维持磷酸盐的体内平衡。
J Clin Invest. 2023 Apr 17;133(8):e164610. doi: 10.1172/JCI164610.
4
Emerging Antiarrhythmic Drugs for Atrial Fibrillation.新型抗心律失常药物治疗心房颤动。
Int J Mol Sci. 2022 Apr 7;23(8):4096. doi: 10.3390/ijms23084096.
5
Oxidative stress: a bystander or a causal contributor to atrial remodelling and fibrillation?氧化应激:是旁观者还是心房重构及颤动的因果促成因素?
Cardiovasc Res. 2021 Sep 28;117(11):2291-2293. doi: 10.1093/cvr/cvab124.
6
Molecular Basis of Atrial Fibrillation Pathophysiology and Therapy: A Translational Perspective.心房颤动病理生理学和治疗的分子基础:转化视角。
Circ Res. 2020 Jun 19;127(1):51-72. doi: 10.1161/CIRCRESAHA.120.316363. Epub 2020 Jun 18.
7
Multiscale Coupling of an Agent-Based Model of Tissue Fibrosis and a Logic-Based Model of Intracellular Signaling.基于主体的组织纤维化模型与基于逻辑的细胞内信号传导模型的多尺度耦合
Front Physiol. 2019 Dec 17;10:1481. doi: 10.3389/fphys.2019.01481. eCollection 2019.
8
Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca/Calmodulin-Dependent Protein Kinase II δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling.钙/钙调蛋白依赖性蛋白激酶 II δ 信号通路通过心肌细胞对压力超负荷的反应引发的炎症和 NLRP3 炎性小体激活对于心脏不良重构是必需的。
Circulation. 2018 Nov 27;138(22):2530-2544. doi: 10.1161/CIRCULATIONAHA.118.034621.
9
Enhanced Cardiomyocyte NLRP3 Inflammasome Signaling Promotes Atrial Fibrillation.增强的心肌细胞 NLRP3 炎性小体信号促进心房颤动。
Circulation. 2018 Nov 13;138(20):2227-2242. doi: 10.1161/CIRCULATIONAHA.118.035202.
10
Epac-induced ryanodine receptor type 2 activation inhibits sodium currents in atrial and ventricular murine cardiomyocytes.Epac 诱导的兰尼碱受体 2 激活抑制心房和心室鼠心肌细胞中的钠电流。
Clin Exp Pharmacol Physiol. 2018 Mar;45(3):278-292. doi: 10.1111/1440-1681.12870. Epub 2017 Dec 7.

Increases in dietary phosphate levels can augment atrial arrhythmias.

作者信息

Ganesan P, Narayan S M, Giles W R

机构信息

Department of Medicine, Stanford University, Palo Alto, California, USA.

Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta, Canada.

出版信息

Acta Physiol (Oxf). 2023 Jun;238(2):e13978. doi: 10.1111/apha.13978. Epub 2023 Apr 30.

DOI:10.1111/apha.13978
PMID:37062019
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11042787/
Abstract
摘要