Fuher Alexandra Nicole, Borne Ryan, Cunningham John
University of Colorado Anschutz Medical Campus, Department of Internal Medicine, Aurora, Colorado.
University of Colorado Health, Division of Cardiology, Colorado Springs, Colorado.
Clin Pract Cases Emerg Med. 2023 May;7(2):106-109. doi: 10.5811/cpcem.1413.
Late atrial arrhythmias after catheter ablation for atrial fibrillation occur in up to 30% of post-ablation patients and are increasingly encountered by emergency physicians. However, diagnosing the exact mechanism of the arrhythmia on the surface electrocardiogram (ECG) remains challenging due to atrial scarring leading to heterogeneous P-wave morphology.
A 74-year-old male with a history of prior catheter ablation for atrial fibrillation presented with palpitations and subacute symptoms of heart failure. The patient's ECG revealed narrow complex tachycardia with more P waves than QRS complexes. The differential diagnosis included typical flutter, atypical flutter, and focal atrial tachycardias with 2:1 conduction block. P waves were positive in V1 and across all precordial leads (absent precordial transition). This favors atypical flutter originating from the left atrium over typical cavotricuspid isthmus-dependent right atrial flutter. Transthoracic echocardiogram showed a reduced ejection fraction due to tachycardia-mediated cardiomyopathy. The patient underwent a repeat electrophysiology study and ablation, which confirmed the presence of an atypical flutter circuit using the mitral annulus, known as perimitral flutter. Repeat catheter ablation resulted in maintenance of sinus rhythm. At follow-up, his ejection fraction recovered.
Recognizing ECG findings suggestive of atypical flutter impacts initial emergency department decisions and triage as atypical flutter post-atrial fibrillation ablation is frequently resistant to rate-controlling medications and often requires cardiology and/or electrophysiology consultation if available.
房颤导管消融术后晚期房性心律失常在高达30%的消融后患者中出现,急诊科医生越来越多地遇到此类情况。然而,由于心房瘢痕导致P波形态不均一,在体表心电图(ECG)上诊断心律失常的确切机制仍然具有挑战性。
一名74岁男性,有房颤导管消融病史,出现心悸和心力衰竭亚急性症状。患者的心电图显示窄QRS波心动过速,P波多于QRS波群。鉴别诊断包括典型房扑、非典型房扑和伴有2:1传导阻滞的局灶性房性心动过速。V1导联及所有胸前导联P波均为正向(胸前导联无过渡区)。这支持起源于左心房的非典型房扑,而非典型三尖瓣峡部依赖性右心房房扑。经胸超声心动图显示因心动过速介导的心肌病导致射血分数降低。患者接受了重复电生理检查和消融,证实存在一种利用二尖瓣环的非典型房扑环路,即二尖瓣环周围房扑。重复导管消融使窦性心律得以维持。随访时,他的射血分数恢复。
认识到提示非典型房扑的心电图表现会影响急诊科的初始决策和分诊,因为房颤消融术后的非典型房扑通常对心率控制药物耐药,如果可能的话,往往需要心内科和/或电生理会诊。