• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

相似文献

1
Inability of chlorophenols to induce 6-thioguanine-resistant mutants in V79 Chinese hamster cells.氯酚类物质无法在V79中国仓鼠细胞中诱导出对6-硫代鸟嘌呤具有抗性的突变体。
Mutat Res. 1986 Aug-Sep;171(2-3):165-8. doi: 10.1016/0165-1218(86)90050-9.
2
Genotoxicity of 2,4,6-trichlorophenol in V79 Chinese hamster cells.2,4,6-三氯苯酚对V79中国仓鼠细胞的遗传毒性
Mutat Res. 1992;280(3):175-9. doi: 10.1016/0165-1218(92)90046-3.
3
Expression time for benzo[a]pyrene-induced 6-thioguanine-resistant mutations in V79 Chinese hamster cells.苯并[a]芘诱导的V79中国仓鼠细胞中6-硫鸟嘌呤抗性突变的表达时间。
Mutat Res. 1982 Aug;95(2-3):353-62. doi: 10.1016/0027-5107(82)90270-6.
4
Inability of diethylstilbestrol to induce 6-thioguanine-resistant mutants and to inhibit metabolic cooperation of V79 Chinese hamster cells.
Mutat Res. 1983 Feb;107(2):427-32. doi: 10.1016/0027-5107(83)90181-1.
5
Lack of induction of sister chromatid exchanges and of mutation to 6-thioguanine resistance in V79 cells by butylated hydroxyanisole with and without activation by rat or hamster hepatocytes.在有或无大鼠或仓鼠肝细胞激活的情况下，丁基化羟基茴香醚未诱导V79细胞发生姐妹染色单体交换以及对6-硫鸟嘌呤产生抗性突变。
Cancer Lett. 1985 May;27(1):61-9. doi: 10.1016/0304-3835(85)90009-6.
6
Mutagenic activity of 3 diazoacetylglycine derivatives on V79 Chinese hamster cells.3-重氮乙酰甘氨酸衍生物对V79中国仓鼠细胞的诱变活性。
Mutat Res. 1980 Aug;78(4):375-9. doi: 10.1016/0165-1218(80)90044-0.
7
Cytotoxic, and mutagenic effects at the HGPRT locus of 4CMB in V79 Chinese hamster cells.4-氯-3-甲基苯硼酸（4CMB）对V79中国仓鼠细胞次黄嘌呤鸟嘌呤磷酸核糖转移酶（HGPRT）位点的细胞毒性和诱变作用。
Mutat Res. 1982 Jan-Feb;100(1-4):235-8. doi: 10.1016/0165-1218(82)90052-0.
8
Induction of temperature-sensitive 6-thioguanine-resistant mutants as a measure of base-pair substitution mutations in Chinese hamster ovary cells.
Mutat Res. 1984 Feb;139(2):95-100. doi: 10.1016/0165-7992(84)90110-6.
9
Cytotoxic and genotoxic properties of tert.-butyl-p-quinone (TBQ) in an in vitro assay system with Chinese hamster V79 cells and in strain D7 of Saccharomyces cerevisiae.叔丁基对苯二酚（TBQ）在中国仓鼠V79细胞体外检测系统及酿酒酵母D7菌株中的细胞毒性和遗传毒性特性
Mutat Res. 1993 Mar;299(1):9-18. doi: 10.1016/0165-1218(93)90113-r.
10
Induction of mutation in V79 Chinese hamster cells by tetrachlorohydroquinone, a metabolite of pentachlorophenol.
Mutat Res. 1991 May;260(1):83-7. doi: 10.1016/0165-1218(91)90083-x.

引用本文的文献

1
Evidence of apoptosis in some cell types due to pentachlorophenol (PCP) in Heteropneustes fossilis.证明五氯苯酚（PCP）会导致褐牙鲆某些细胞类型发生细胞凋亡。
Saudi J Biol Sci. 2013 Jan;20(1):45-9. doi: 10.1016/j.sjbs.2012.10.002. Epub 2012 Oct 27.
2
Estimation of selected phenols in drinking water with in situ acetylation and study on the DNA damaging properties of polychlorinated phenols.
Arch Environ Contam Toxicol. 1995 May;28(4):537-42. doi: 10.1007/BF00211639.

氯酚类物质无法在V79中国仓鼠细胞中诱导出对6-硫代鸟嘌呤具有抗性的突变体。

Inability of chlorophenols to induce 6-thioguanine-resistant mutants in V79 Chinese hamster cells.

作者信息

Jansson K, Jansson V

出版信息

Mutat Res. 1986 Aug-Sep;171(2-3):165-8. doi: 10.1016/0165-1218(86)90050-9.

DOI:10.1016/0165-1218(86)90050-9
PMID:3748065
Abstract

The induction of mutation at the hypoxanthine-guanine phosphoribosyl transferase locus and cytotoxicities of 6 different chlorophenols (2,4- and 2,6-dichlorophenol, 2,4,5- and 2,4,6-trichlorophenol, 2,3,4,6-tetrachlorophenol and pentachlorophenol) were examined in V79 Chinese hamster cells without exogenous metabolic activation. The chlorophenols were cytotoxic to V79 cells, but failed to produce significant increases in the frequency of 6-thioguanine-resistant mutants.

摘要

在无外源性代谢活化的情况下，研究了6种不同氯酚（2,4-二氯酚、2,6-二氯酚、2,4,5-三氯酚、2,4,6-三氯酚、2,3,4,6-四氯酚和五氯酚）对V79中国仓鼠细胞次黄嘌呤-鸟嘌呤磷酸核糖转移酶位点的诱变作用及细胞毒性。氯酚对V79细胞具有细胞毒性，但未能使6-硫鸟嘌呤抗性突变体的频率显著增加。