Endothelin Signaling Mediates Biliary-Endothelial Crosstalk in Primary Sclerosing Cholangitis.
作者信息
Yin Chunyue
机构信息
Division of Gastroenterology, Hepatology and Nutrition, Division of Developmental Biology, Department of Pediatrics, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio.
出版信息
Cell Mol Gastroenterol Hepatol. 2023;16(4):643-645. doi: 10.1016/j.jcmgh.2023.07.003. Epub 2023 Jul 28.
Abstract
摘要
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本文引用的文献
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Endothelin Receptor-A Inhibition Decreases Ductular Reaction, Liver Fibrosis, and Angiogenesis in a Model of Cholangitis.内皮素受体-A 抑制可减少胆管炎模型中的胆小管反应、肝纤维化和血管生成。
Cell Mol Gastroenterol Hepatol. 2023;16(4):513-540. doi: 10.1016/j.jcmgh.2023.06.005. Epub 2023 Jun 17.
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Endothelin receptor B enhances liver injury and pro-inflammatory responses by increasing G-protein-coupled receptor kinase-2 expression in primary biliary cholangitis.内皮素受体 B 通过增加原发性胆汁性胆管炎中 G 蛋白偶联受体激酶-2 的表达来增强肝损伤和促炎反应。
Sci Rep. 2022 Nov 17;12(1):19772. doi: 10.1038/s41598-022-21816-x.
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Recent discoveries in microbiota dysbiosis, cholangiocytic factors, and models for studying the pathogenesis of primary sclerosing cholangitis.微生物群失调、胆管细胞因子及原发性硬化性胆管炎发病机制研究模型的最新发现
Open Med (Wars). 2022 May 13;17(1):915-929. doi: 10.1515/med-2022-0481. eCollection 2022.
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Ductular reaction promotes intrahepatic angiogenesis through Slit2-Roundabout 1 signaling.小管反应通过 Slit2-Roundabout 1 信号促进肝内血管生成。
Hepatology. 2022 Feb;75(2):353-368. doi: 10.1002/hep.32140. Epub 2021 Dec 15.
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