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红细胞结合蛋白的顺序作用使疟原虫分阶段承诺入侵。

Sequential roles for red blood cell binding proteins enable phased commitment to invasion for malaria parasites.

机构信息

Department of Infection Biology, Faculty of Infectious and Tropical Disease, London School of Hygiene and Tropical Medicine, London, WC1E 7HT, UK.

Department of Pathobiology and Population Sciences, Royal Veterinary College, Hawkshead Lane, Hatfield, AL9 7TA, UK.

出版信息

Nat Commun. 2023 Aug 1;14(1):4619. doi: 10.1038/s41467-023-40357-z.

Abstract

Invasion of red blood cells (RBCs) by Plasmodium merozoites is critical to their continued survival within the host. Two major protein families, the Duffy binding-like proteins (DBPs/EBAs) and the reticulocyte binding like proteins (RBLs/RHs) have been studied extensively in P. falciparum and are hypothesized to have overlapping, but critical roles just prior to host cell entry. The zoonotic malaria parasite, P. knowlesi, has larger invasive merozoites and contains a smaller, less redundant, DBP and RBL repertoire than P. falciparum. One DBP (DBPα) and one RBL, normocyte binding protein Xa (NBPXa) are essential for invasion of human RBCs. Taking advantage of the unique biological features of P. knowlesi and iterative CRISPR-Cas9 genome editing, we determine the precise order of key invasion milestones and demonstrate distinct roles for each family. These distinct roles support a mechanism for phased commitment to invasion and can be targeted synergistically with invasion inhibitory antibodies.

摘要

疟原虫裂殖子入侵红细胞对其在宿主体内的持续生存至关重要。两个主要的蛋白质家族,即达夫结合蛋白样蛋白(DBPs/EBAs)和网织红细胞结合蛋白样蛋白(RBLs/RHs),在恶性疟原虫中得到了广泛的研究,并假设它们在进入宿主细胞之前具有重叠但关键的作用。人畜共患疟原虫——嗜人疟原虫具有更大的入侵裂殖子,并且含有比恶性疟原虫更小、更不冗余的 DBP 和 RBL 库。一个 DBP(DBPα)和一个 RBL,即正常红细胞结合蛋白 Xa(NBPXa),对于入侵人类红细胞是必不可少的。利用嗜人疟原虫独特的生物学特征和迭代的 CRISPR-Cas9 基因组编辑,我们确定了关键入侵里程碑的精确顺序,并证明了每个家族的独特作用。这些不同的作用支持了分阶段入侵的机制,并可以与入侵抑制抗体协同作用。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ac82/10393984/047e7676e712/41467_2023_40357_Fig1_HTML.jpg

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