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The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms.

作者信息

Hunter D R, Haworth R A

出版信息

Arch Biochem Biophys. 1979 Jul;195(2):453-9. doi: 10.1016/0003-9861(79)90371-0.

DOI:10.1016/0003-9861(79)90371-0
PMID:383019
Abstract
摘要

相似文献

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The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms.线粒体中钙离子诱导的膜转变。I. 保护机制。
Arch Biochem Biophys. 1979 Jul;195(2):453-9. doi: 10.1016/0003-9861(79)90371-0.
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The Ca2+-induced membrane transition in mitochondria. III. Transitional Ca2+ release.线粒体中Ca2+诱导的膜转变。III. 过渡性Ca2+释放。
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The Ca2+-induced membrane transition in mitochondria. II. Nature of the Ca2+ trigger site.
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Duramycin effects on the structure and function of heart mitochondria. I. Structural alterations and changes in membrane permeability.
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Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adenine nucleotide translocase.环孢素抑制Ca2+诱导的肝脏和心脏线粒体大幅度肿胀，可能是由于该抑制剂与线粒体基质肽基脯氨酰顺反异构酶结合，阻止其与腺嘌呤核苷酸转位酶相互作用所致。
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Isolation of functional membrane proteins related to or identical with the ADP, ATP carrier of mitochondria.与线粒体ADP、ATP载体相关或相同的功能性膜蛋白的分离。
Methods Enzymol. 1979;56:407-14. doi: 10.1016/0076-6879(79)56039-x.
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Calcium-dependent opening of a non-specific pore in the mitochondrial inner membrane is inhibited at pH values below 7. Implications for the protective effect of low pH against chemical and hypoxic cell damage.线粒体内膜中一种非特异性孔道的钙依赖性开放在pH值低于7时受到抑制。这对低pH值对化学和缺氧性细胞损伤的保护作用具有启示意义。
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Exploration of nucleotide binding sites in the mitochondrial membrane by 2-azido-[alpha-32P]ADP.利用2-叠氮基-[α-32P]ADP对线粒体膜中的核苷酸结合位点进行探索。
FEBS Lett. 1985 Jan 28;180(2):212-8. doi: 10.1016/0014-5793(85)81073-5.

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