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sl和w位点突变小鼠中的嗜酸性粒细胞缺乏症。

Eosinophilic granulocyte deficiency in mice mutant in sl and w loci.

作者信息

Wiktor-Jedrzejczak W, Ratajczak M

出版信息

Experientia. 1985 Dec 15;41(12):1596-8. doi: 10.1007/BF01964826.

DOI:10.1007/BF01964826
PMID:3841073
Abstract

Homozygous Sl/Sl and Wv/Wv mice were found to have approximately 15% of the normal number of circulating eosinophils. Furthermore, these mice exhibited reduced numbers of eosinophilic granulocytes in the bone marrow, spleen and thymus as compared to littermate control normal mice.

摘要

纯合子Sl/Sl和Wv/Wv小鼠的循环嗜酸性粒细胞数量约为正常数量的15%。此外,与同窝对照正常小鼠相比,这些小鼠骨髓、脾脏和胸腺中的嗜酸性粒细胞数量减少。

相似文献

1
Eosinophilic granulocyte deficiency in mice mutant in sl and w loci.sl和w位点突变小鼠中的嗜酸性粒细胞缺乏症。
Experientia. 1985 Dec 15;41(12):1596-8. doi: 10.1007/BF01964826.
2
Reconstitution of the W/Wv stem cell differentiation defect by infection with Rauscher leukemia virus.通过劳氏肉瘤病毒感染重建W/Wv干细胞分化缺陷。
J Natl Cancer Inst. 1985 Aug;75(2):361-8.
3
An autopsy case of lymphopenic hypogammaglobulinemia.
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4
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Res Vet Sci. 1977 Sep;23(2):196-203.
5
Compensatory mechanisms in platelet production: lack of a paracrine response in W/Wv mice treated with 5-fluorouracil.血小板生成中的代偿机制:5-氟尿嘧啶处理的W/Wv小鼠中旁分泌反应的缺失。
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6
Organ interactions in the regulation of hematopoiesis: in vitro interactions of bone, thymus, and spleen with bone marrow stem cells in normal, Sl/Sld and W/Wv mice.造血调控中的器官相互作用:正常、Sl/Sld和W/Wv小鼠中骨、胸腺和脾脏与骨髓干细胞的体外相互作用
J Cell Physiol. 1977 Jan;90(1):105-15. doi: 10.1002/jcp.1040900113.
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Subacute toxicity of dietary T-2 toxin in mice: morphological and hematological effects.日粮中T-2毒素对小鼠的亚急性毒性:形态学和血液学影响
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Histo-pathologic observations of the nervous and lymphoid tissues of "lethargic" mutant mice.“嗜睡”突变小鼠神经和淋巴组织的组织病理学观察
Tex Rep Biol Med. 1972 Spring;30(1):23-39.
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Susceptibility of multipotent haemopoietic stem cell deficient W/Wv mice to Plasmodium berghei-infection.多能造血干细胞缺陷型W/Wv小鼠对伯氏疟原虫感染的易感性。
Immunol Cell Biol. 1991 Oct;69 ( Pt 5):355-60. doi: 10.1038/icb.1991.51.
10
The mast cell-committed progenitor. II. W/Wv mice do not make mast cell-committed progenitors and S1/S1d fibroblasts do not support development of normal mast cell-committed progenitors.肥大细胞定向祖细胞。II. W/Wv小鼠不产生肥大细胞定向祖细胞,且S1/S1d成纤维细胞不支持正常肥大细胞定向祖细胞的发育。
J Immunol. 1989 Apr 1;142(7):2418-23.

本文引用的文献

1
A backcross method to prevent prenatal death of steel mutant mice.一种预防钢铁突变小鼠产前死亡的回交方法。
J Hered. 1984 Jan-Feb;75(1):73. doi: 10.1093/oxfordjournals.jhered.a109871.
2
Decreased neutrophils and megakaryocytes in anemic mice of genotype W/W.基因型为W/W的贫血小鼠中性粒细胞和巨核细胞减少。
J Cell Physiol. 1969 Feb;73(1):25-30. doi: 10.1002/jcp.1040730104.
3
Abnormalities of megakaryocytes in S1-S1d mice.S1-S1d小鼠中巨核细胞的异常情况。
Blood. 1973 Dec;42(6):865-71.
4
Reduced blood and marrow neutrophils and granulocytic colony-forming cells in S1/S1d mice.S1/S1d小鼠的血液和骨髓中性粒细胞以及粒细胞集落形成细胞减少。
Proc Soc Exp Biol Med. 1976 Jul;152(3):398-402. doi: 10.3181/00379727-152-39405.
5
Immune system of the W/Wv mice: functional studies.W/Wv小鼠的免疫系统:功能研究。
Bull Acad Pol Sci Biol. 1979;27(11):895-901.
6
Hereditary anemias of the mouse: a review for geneticists.
Adv Genet. 1979;20:357-459.
7
Decreased production of mast cells in S1/S1d anemic mice.S1/S1d贫血小鼠中肥大细胞的产生减少。
Blood. 1979 Mar;53(3):492-7.