Godleski Stephanie, Schuetze Pamela, Eiden Rina D, Nickerson Amanda B, Ostrov Jamie M
Department of Psychology, College of Liberal Arts, Rochester Institute of Technology.
Department of Psychology, State University of New York Buffalo State.
J Appl Dev Psychol. 2022 Nov-Dec;83. doi: 10.1016/j.appdev.2022.101474. Epub 2022 Nov 8.
Few studies have examined etiological pathways from prenatal substance exposure to adolescent reactive aggression. We tested a conceptual model that included hypothesized pathways from prenatal substance exposure to adolescent aggression via autonomic reactivity and violence exposure from infancy to early school age and maternal harshness across early childhood. The sample included 216 families (106 boys) who primarily self-identified as Black or Mixed Race. Results supported the hypothesized path from violence exposure across early childhood and early school age to school age autonomic reactivity and early adolescent reactive aggression. There was also a significant interaction effect of sympathetic and parasympathetic reactivity on adolescent reactive aggression, with sympathetic arousal and parasympathetic suppression at early school age associated with higher reactive relational and physical aggression in adolescence. Results emphasize the importance of early experiences and autonomic nervous system changes in contributing to the cascade of risk for reactive aggression in early adolescence.
很少有研究探讨从产前物质暴露到青少年反应性攻击行为的病因途径。我们测试了一个概念模型，该模型包括从产前物质暴露到青少年攻击行为的假设途径，即通过自主反应性以及从婴儿期到学龄早期的暴力暴露和整个幼儿期的母亲严厉程度。样本包括216个家庭（106名男孩），他们主要自我认定为黑人或混血儿。结果支持了从幼儿期和学龄早期的暴力暴露到学龄期自主反应性和青少年早期反应性攻击行为的假设路径。交感神经和副交感神经反应性对青少年反应性攻击行为也有显著的交互作用，学龄早期的交感神经唤醒和副交感神经抑制与青少年期更高的反应性关系攻击和身体攻击有关。结果强调了早期经历和自主神经系统变化在导致青少年早期反应性攻击行为风险级联中的重要性。
