巨噬细胞激活以实现肿瘤细胞毒性：中间反应分析 - Suppr

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Macrophage activation for tumor cytotoxicity: analysis of intermediary reactions.

作者信息

Meltzer M S, Ruco L P, Boraschi D, Nacy C A

出版信息

J Reticuloendothel Soc. 1979 Oct;26(4):403-15.

PMID:390144

Abstract

摘要

相似文献

Macrophage activation for tumor cytotoxicity: analysis of intermediary reactions.巨噬细胞激活以实现肿瘤细胞毒性：中间反应分析

J Reticuloendothel Soc. 1979 Oct;26(4):403-15.

Macrophage activation for tumor cytotoxicity: development of macrophage cytotoxic activity requires completion of a sequence of short-lived intermediary reactions.

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Defective tumoricidal capacity of macrophages from A/J mice. II. Comparison of the macrophage cytotoxic defect of A/J mice with that of lipid A-unresponsive C3H/HeJ mice.A/J小鼠巨噬细胞的肿瘤杀伤能力缺陷。II. A/J小鼠巨噬细胞细胞毒性缺陷与脂质A无反应性C3H/HeJ小鼠的比较。

J Immunol. 1979 Apr;122(4):1592-97.

Macrophage activation for tumor cytotoxicity: mechanisms of macrophage activation by lymphokines.

Adv Exp Med Biol. 1979;121B:381-98. doi: 10.1007/978-1-4684-8914-9_36.

Genetic aspects of macrophage activation for tumor cytotoxicity.巨噬细胞激活以产生肿瘤细胞毒性的遗传学方面。

Adv Exp Med Biol. 1982;141:119-28. doi: 10.1007/978-1-4684-8088-7_12.

Macrophage activation for tumor cytotoxicity: control of macrophage tumoricidal capacity by the LPS gene.巨噬细胞激活以实现肿瘤细胞毒性：LPS基因对巨噬细胞杀瘤能力的控制

J Immunol. 1978 Aug;121(2):543-8.

Defective tumoricidal capacity of macrophages from P/J mice: characterization of the macrophage cytotoxic defect after in vivo and in vitro activation stimuli.P/J 小鼠巨噬细胞的肿瘤杀伤能力缺陷：体内和体外激活刺激后巨噬细胞细胞毒性缺陷的特征

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Defective tumoricidal capacity of macrophages from P/J mice: tumoricidal defect involves abnormalities in lymphokine-derived activation stimuli and in mononuclear phagocyte responsiveness.P/J小鼠巨噬细胞的肿瘤杀伤能力缺陷：肿瘤杀伤缺陷涉及淋巴因子衍生的激活刺激和单核吞噬细胞反应性的异常。

J Immunol. 1980 Aug;125(2):777-82.

Macrophage activation for tumor cytotoxicity: increased lymphokine responsiveness of peritoneal macrophages during acute inflammation.巨噬细胞激活以产生肿瘤细胞毒性：急性炎症期间腹膜巨噬细胞对淋巴因子的反应性增强。

J Immunol. 1978 Mar;120(3):1054-62.

Modulation of the tumoricidal function of activated macrophages by bacterial endotoxin and mammalian macrophage activation factor (s).

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Quantitative studies of natural immunity to solid tumours in rats. The nature of the killer cell depends on the type of assay.大鼠对实体瘤天然免疫的定量研究。杀伤细胞的性质取决于检测方法的类型。

Immunology. 1980 Nov;41(3):673-80.

Susceptibility differences of inbred strains of mice to blastomycosis.

Infect Immun. 1981 Apr;32(1):160-8. doi: 10.1128/iai.32.1.160-168.1981.

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Histochem J. 1982 Mar;14(2):221-37. doi: 10.1007/BF01041217.

Fate of Listeria monocytogenes in resident and activated macrophages.单核细胞增生李斯特菌在驻留巨噬细胞和活化巨噬细胞中的命运。

Infect Immun. 1981 Jul;33(1):11-6. doi: 10.1128/iai.33.1.11-16.1981.

Macrophages in resistance to rickettsial infections: early host defense mechanisms in experimental scrub typhus.巨噬细胞在抵抗立克次体感染中的作用：实验性恙虫病的早期宿主防御机制

Infect Immun. 1981 Mar;31(3):1239-50. doi: 10.1128/iai.31.3.1239-1250.1981.

Effect of acute nutritional deprivation on immune function in mice. I. Macrophages.急性营养缺乏对小鼠免疫功能的影响。I. 巨噬细胞。

Immunology. 1983 Mar;48(3):543-50.

Interferon decreases production of hydrogen peroxide by macrophages: correlation with reduction of suppressive capacity and of anti-microbial activity.干扰素可降低巨噬细胞过氧化氢的产生：与抑制能力和抗菌活性降低相关。

Immunology. 1983 Nov;50(3):359-68.

Macrophage function in Mycobacterium lepraemurium infection: morphological and functional changes of peritoneal and splenic macrophages in vitro.鼠麻风分枝杆菌感染中的巨噬细胞功能：体外腹腔和脾脏巨噬细胞的形态学及功能变化

Infect Immun. 1983 Jan;39(1):353-61. doi: 10.1128/iai.39.1.353-361.1983.

Detection of either rapidly cytolytic macrophages or NK cells in "activated" peritoneal exudates depends on the method of analysis and the target cell type.在“活化的”腹腔渗出物中检测快速溶细胞性巨噬细胞或自然杀伤细胞取决于分析方法和靶细胞类型。

Immunology. 1981 Apr;42(4):561-8.

Interaction of high or low metastatic related tumor lines with normal or lymphokine-activated syngeneic peritoneal macrophages: in vitro analysis of tumor cell binding and cytostasis.高转移或低转移相关肿瘤细胞系与正常或淋巴因子激活的同基因腹膜巨噬细胞的相互作用：肿瘤细胞结合及细胞生长抑制的体外分析

Clin Exp Metastasis. 1985 Jan-Mar;3(1):29-43. doi: 10.1007/BF01758952.

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