淀粉样变性的主动调节 - Suppr

Active regulation of amyloidosis.

作者信息

Lin Yi

机构信息

School of Life Sciences, Tsinghua-Peking Joint Centre for Life Sciences, IDG/McGovern Institute for Brain Research, State Key Laboratory of Membrane Biology, Tsinghua University, Beijing 100084, China.

出版信息

Proc Natl Acad Sci U S A. 2024 Aug 6;121(32):e2409665121. doi: 10.1073/pnas.2409665121. Epub 2024 Jul 29.

DOI:10.1073/pnas.2409665121

PMID:39074293

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11317571/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3642/11317571/5eca3b06e03e/pnas.2409665121fig01.jpg

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Active regulation of amyloidosis.

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It's not just a phase: function and characteristics of RNA-binding proteins in phase separation.

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RNA-binding protein FXR1 is presented in rat brain in amyloid form.

Sci Rep. 2019 Dec 12;9(1):18983. doi: 10.1038/s41598-019-55528-6.

CPEB3 inhibits translation of mRNA targets by localizing them to P bodies.

Proc Natl Acad Sci U S A. 2019 Sep 3;116(36):18078-18087. doi: 10.1073/pnas.1815275116. Epub 2019 Aug 15.

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MAVS forms functional prion-like aggregates to activate and propagate antiviral innate immune response.

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Science. 1899 Jul 14;10(237):33-45. doi: 10.1126/science.10.237.33.

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Active regulation of amyloidosis.

Proc Natl Acad Sci U S A. 2024 Aug 6;121(32):e2409665121. doi: 10.1073/pnas.2409665121. Epub 2024 Jul 29.

[Cardiac manifestations of amyloidosis].

Arkh Patol. 2009 Mar-Apr;71(2):3-6.

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Nihon Rinsho. 1979 Sep 10;37(9):3245-58.

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Amyloid. 2017 Mar;24(sup1):130-131. doi: 10.1080/13506129.2017.1296422.

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Amyloid. 2017 Mar;24(sup1):171-172. doi: 10.1080/13506129.2017.1295948.

[Amyloidosis and hepatic amyloidosis].

Nihon Rinsho. 1993 Feb;51(2):453-7.

[Amyloidosis associated with aging].

Nihon Rinsho. 1991 Apr;49(4):814-20.

本文引用的文献

RNA tailing machinery drives amyloidogenic phase transition.

Proc Natl Acad Sci U S A. 2024 Jun 4;121(23):e2316734121. doi: 10.1073/pnas.2316734121. Epub 2024 May 28.

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Stress-Induced Low Complexity RNA Activates Physiological Amyloidogenesis.

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Active regulation of amyloidosis.

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