Do , malnutrition, and long COVID-19 compound the risk factors for stroke in adverse environments?
作者信息
Buzaglo Alexander Vasconcelos, Vieira Carlos Meton de Alencar Gadelha, Ciurleo Gabriella Cunha Vieira, Belayev Ludmila, Oriá Reinaldo B
机构信息
Laboratory of the Biology of Tissue Healing, Ontogeny and Nutrition, Department of Morphology and Institute of Biomedicine, School of Medicine, Federal University of Ceara, Fortaleza, Ceará, Brazil.
School of Medicine, Neuroscience Center of Excellence, Louisiana State University Health Sciences Center, New Orleans, LA, United States.
出版信息
Front Nutr. 2024 Jul 23;11:1422218. doi: 10.3389/fnut.2024.1422218. eCollection 2024.
Abstract
摘要
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Do , malnutrition, and long COVID-19 compound the risk factors for stroke in adverse environments?在不利环境中，痴呆、营养不良和长期新冠病毒感染是否会增加中风的风险因素？
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本文引用的文献
1
The Clinical Aspects of COVID and Alzheimer's Disease: A Round-Up of Where Things Stand and Are Headed.COVID 和阿尔茨海默病的临床方面：现状和未来方向综述。
J Alzheimers Dis. 2024;99(4):1159-1171. doi: 10.3233/JAD-231368.
2
Association between abnormal lipid metabolism and Alzheimer's disease: New research has revealed significant findings on the APOE4 genotype in microglia.异常脂质代谢与阿尔茨海默病的关系：新的研究揭示了载脂蛋白 E4 基因型在小胶质细胞中的重要发现。
Biosci Trends. 2024 Jun 6;18(2):195-197. doi: 10.5582/bst.2024.01092. Epub 2024 Apr 17.
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Prevalence and risk factors for persistent symptoms after COVID-19: a systematic review and meta-analysis.新冠病毒病后持续症状的患病率及危险因素：一项系统评价与荟萃分析
Clin Microbiol Infect. 2024 Mar;30(3):328-335. doi: 10.1016/j.cmi.2023.10.016. Epub 2023 Oct 20.
4
Do 4 and long COVID-19 increase the risk for neurodegenerative diseases in adverse environments and poverty?4 型糖尿病和长期新冠病毒感染是否会在不利环境和贫困状况下增加患神经退行性疾病的风险？
Front Neurosci. 2023 Aug 24;17:1229073. doi: 10.3389/fnins.2023.1229073. eCollection 2023.
5
Pathogenic mechanisms of post-acute sequelae of SARS-CoV-2 infection (PASC).SARS-CoV-2 感染后后遗症（PASC）的发病机制。
Elife. 2023 Mar 22;12:e86002. doi: 10.7554/eLife.86002.
6
ApoE4 associated with severe COVID-19 outcomes via downregulation of ACE2 and imbalanced RAS pathway.载脂蛋白 E4 通过下调 ACE2 和失衡的 RAS 通路与严重的 COVID-19 结局相关。
J Transl Med. 2023 Feb 9;21(1):103. doi: 10.1186/s12967-023-03945-7.
7
The Epidemiology of Long Coronavirus Disease in US Adults.美国成年人长冠状病毒病的流行病学。
Clin Infect Dis. 2023 May 3;76(9):1636-1645. doi: 10.1093/cid/ciac961.
8
Blood-based biomarkers for diagnosis, prognosis, and severity prediction of COVID-19: Opportunities and challenges.用于COVID-19诊断、预后和严重程度预测的血液生物标志物：机遇与挑战。
J Family Med Prim Care. 2022 Aug;11(8):4330-4341. doi: 10.4103/jfmpc.jfmpc_2283_21. Epub 2022 Aug 30.
9
A "multi-omics" analysis of blood-brain barrier and synaptic dysfunction in APOE4 mice.载脂蛋白 E4 小鼠血脑屏障和突触功能障碍的“多组学”分析。
J Exp Med. 2022 Nov 7;219(11). doi: 10.1084/jem.20221137. Epub 2022 Aug 30.
10
APOE interacts with ACE2 inhibiting SARS-CoV-2 cellular entry and inflammation in COVID-19 patients.载脂蛋白 E（APOE）与血管紧张素转换酶 2（ACE2）相互作用，抑制 COVID-19 患者体内的 SARS-CoV-2 细胞进入和炎症反应。
Signal Transduct Target Ther. 2022 Aug 1;7(1):261. doi: 10.1038/s41392-022-01118-4.
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