Kakadiya Jay, Varsadiya Kush, Kakadiya Chintan, Prajapati Maulik, Patel Pritesh
Department of Internal Medicine, Government Medical College, Surat, Surat, IND.
Department of Internal Medicine, Shri M. P. (Meghaji Pethraj) Shah Government Medical College, Jamnagar, IND.
Cureus. 2024 Jul 27;16(7):e65489. doi: 10.7759/cureus.65489. eCollection 2024 Jul.
Dengue fever is a viral hemorrhagic fever mainly transmitted by mosquitoes and is especially prevalent in equatorial regions. The presentation of dengue fever can range from mild symptoms, such as fever and body aches, to severe symptoms, such as hemorrhagic bleeding and shock. Although it is a non-neurotropic virus, it rarely manifests as a neurological abnormality. Previous data suggests that the incidence of electrolyte disturbance is increasing in patients with dengue. Here, we have described a case of dengue fever with hypokalemia and renal glycosuria. Studies show that the probable mechanism of developing hypokalemia is increased insulin and catecholamine, but it is still not well-established. We propose a mechanism that can explain both hypokalemia and renal glycosuria in our case.
登革热是一种主要由蚊子传播的病毒性出血热，在赤道地区尤为普遍。登革热的表现范围从轻微症状，如发热和身体疼痛，到严重症状，如出血和休克。虽然它是一种非嗜神经性病毒，但很少表现为神经异常。先前的数据表明，登革热患者电解质紊乱的发生率正在增加。在此，我们描述了一例伴有低钾血症和肾性糖尿的登革热病例。研究表明，发生低钾血症的可能机制是胰岛素和儿茶酚胺增加，但仍未完全明确。我们提出了一种机制，可以解释我们病例中的低钾血症和肾性糖尿。
