• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

氧化还原失衡与肾脏病中的线粒体异常 - 第二卷。

Redox Imbalance and Mitochondrial Abnormalities in Kidney Disease-Volume II.

机构信息

Department of Pharmaceutical Sciences, UNT System College of Pharmacy, University of North Texas Health Science Center, Fort Worth, TX 76107, USA.

Institute of Medicinal Biotechnology, Chinese Academy of Medical Science and Peking Union Medical College, Beijing 100050, China.

出版信息

Biomolecules. 2024 Aug 9;14(8):973. doi: 10.3390/biom14080973.

DOI:10.3390/biom14080973
PMID:39199361
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11353165/
Abstract

The kidney performs fundamental functions by eliminating metabolic waste and reabsorbing essential nutrients and electrolytes such as glucose, proteins, ions, and anions [...].

摘要

肾脏通过排泄代谢废物和重吸收必需的营养物质和电解质,如葡萄糖、蛋白质、离子和阴离子等,来发挥基本功能。

相似文献

1
Redox Imbalance and Mitochondrial Abnormalities in Kidney Disease-Volume II.氧化还原失衡与肾脏病中的线粒体异常 - 第二卷。
Biomolecules. 2024 Aug 9;14(8):973. doi: 10.3390/biom14080973.
2
Redox Imbalance and Mitochondrial Abnormalities in Kidney Disease.氧化还原失衡与肾脏疾病中的线粒体异常。
Biomolecules. 2022 Mar 21;12(3):476. doi: 10.3390/biom12030476.
3
Oxidant Mechanisms in Renal Injury and Disease.肾脏损伤与疾病中的氧化机制
Antioxid Redox Signal. 2016 Jul 20;25(3):119-46. doi: 10.1089/ars.2016.6665. Epub 2016 Apr 26.
4
Artemether ameliorates kidney injury by restoring redox imbalance and improving mitochondrial function in Adriamycin nephropathy in mice.蒿甲醚通过恢复氧化还原失衡和改善阿霉素肾病小鼠的线粒体功能来改善肾脏损伤。
Sci Rep. 2021 Jan 14;11(1):1266. doi: 10.1038/s41598-020-80298-x.
5
Mitochondrial dysfunction in the pathophysiology of renal diseases.肾脏疾病病理生理学中的线粒体功能障碍。
Am J Physiol Renal Physiol. 2014 Feb 15;306(4):F367-78. doi: 10.1152/ajprenal.00571.2013. Epub 2013 Dec 4.
6
Cisplatin-induced nephrotoxicity is associated with oxidative stress, redox state unbalance, impairment of energetic metabolism and apoptosis in rat kidney mitochondria.顺铂诱导的肾毒性与大鼠肾线粒体中的氧化应激、氧化还原状态失衡、能量代谢受损和细胞凋亡有关。
Arch Toxicol. 2007 Jul;81(7):495-504. doi: 10.1007/s00204-006-0173-2. Epub 2007 Jan 11.
7
Mitochondria Damage and Kidney Disease.线粒体损伤与肾脏疾病。
Adv Exp Med Biol. 2017;982:529-551. doi: 10.1007/978-3-319-55330-6_27.
8
The implication of mitochondrial dysfunction and mitochondrial oxidative damage in di (2-ethylhexyl) phthalate induced nephrotoxicity in both and models.线粒体功能障碍和线粒体氧化损伤在邻苯二甲酸二(2-乙基己基)酯诱导的大鼠和小鼠模型肾毒性中的作用。
Toxicol Mech Methods. 2020 Jul;30(6):427-437. doi: 10.1080/15376516.2020.1758980. Epub 2020 Apr 30.
9
Alleviation of renal mitochondrial dysfunction and apoptosis underlies the protective effect of sitagliptin in gentamicin-induced nephrotoxicity.西他列汀对庆大霉素诱导的肾毒性具有保护作用,其机制在于减轻肾线粒体功能障碍和细胞凋亡。
J Pharm Pharmacol. 2016 Apr;68(4):523-32. doi: 10.1111/jphp.12534. Epub 2016 Mar 28.
10
Mitochondrial dysfunction in the pathophysiology of renal diseases.线粒体功能障碍在肾脏疾病病理生理学中的作用
Am J Physiol Renal Physiol. 2024 May 1;326(5):F768-F779. doi: 10.1152/ajprenal.00189.2023. Epub 2024 Mar 7.

引用本文的文献

1
Harnessing Gasotransmitters to Combat Age-Related Oxidative Stress in Smooth Muscle and Endothelial Cells.利用气体信号分子对抗平滑肌和内皮细胞中与年龄相关的氧化应激
Pharmaceuticals (Basel). 2025 Feb 27;18(3):344. doi: 10.3390/ph18030344.

本文引用的文献

1
Renal ischemia/reperfusion induces prominent progressive kidney disease in diabetic mice.肾缺血/再灌注导致糖尿病小鼠发生显著进行性肾脏疾病。
Am J Physiol Endocrinol Metab. 2024 Sep 1;327(3):E302-E312. doi: 10.1152/ajpendo.00237.2023. Epub 2024 Jul 17.
2
The Role of Mitochondrial Sirtuins (SIRT3, SIRT4 and SIRT5) in Renal Cell Metabolism: Implication for Kidney Diseases.线粒体 Sirtuins(SIRT3、SIRT4 和 SIRT5)在肾脏细胞代谢中的作用:对肾脏疾病的影响。
Int J Mol Sci. 2024 Jun 25;25(13):6936. doi: 10.3390/ijms25136936.
3
Inspiring Tactics with the Improvement of Mitophagy and Redox Balance for the Development of Innovative Treatment against Polycystic Kidney Disease.通过提高自噬和氧化还原平衡来激发策略,为多囊肾病的创新治疗开发提供新的思路。
Biomolecules. 2024 Feb 9;14(2):207. doi: 10.3390/biom14020207.
4
Kidney Fibrosis and Oxidative Stress: From Molecular Pathways to New Pharmacological Opportunities.肾纤维化与氧化应激:从分子途径到新的药理学机遇
Biomolecules. 2024 Jan 22;14(1):137. doi: 10.3390/biom14010137.
5
Redox Metabolism and Vascular Calcification in Chronic Kidney Disease.氧化还原代谢与慢性肾脏病血管钙化。
Biomolecules. 2023 Sep 20;13(9):1419. doi: 10.3390/biom13091419.
6
Arginase 2 Promotes Cisplatin-Induced Acute Kidney Injury by the Inflammatory Response of Macrophages.精氨酸酶 2 通过巨噬细胞的炎症反应促进顺铂诱导的急性肾损伤。
Lab Invest. 2023 Oct;103(10):100227. doi: 10.1016/j.labinv.2023.100227. Epub 2023 Aug 2.
7
Renal-Protective Roles of Lipoic Acid in Kidney Disease.硫辛酸在肾脏疾病中的肾脏保护作用。
Nutrients. 2023 Apr 1;15(7):1732. doi: 10.3390/nu15071732.
8
The Combined Administration of Vitamin C and Copper Induces a Systemic Oxidative Stress and Kidney Injury.维生素 C 和铜联合给药诱导全身氧化应激和肾脏损伤。
Biomolecules. 2023 Jan 10;13(1):143. doi: 10.3390/biom13010143.
9
Oxidative Stress and NRF2/KEAP1/ARE Pathway in Diabetic Kidney Disease (DKD): New Perspectives.氧化应激与 NRF2/KEAP1/ARE 通路在糖尿病肾病(DKD)中的作用:新视角。
Biomolecules. 2022 Sep 2;12(9):1227. doi: 10.3390/biom12091227.
10
The Nicotinamide/Streptozotocin Rodent Model of Type 2 Diabetes: Renal Pathophysiology and Redox Imbalance Features.烟酰胺/链脲佐菌素啮齿动物 2 型糖尿病模型:肾脏病理生理学和氧化还原失衡特征。
Biomolecules. 2022 Sep 2;12(9):1225. doi: 10.3390/biom12091225.