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[实验性铜绿假单胞菌败血症病原体从原发性脓毒症病灶侵入血流的结构基础]

[Structural bases for the invasion of the causative agents of experimental Pseudomonas aeruginosa sepsis into the bloodstream from the primary septic focus].

作者信息

Panova N V, Vtiurin B V, Skuba N D, Bochkareva I P

出版信息

Biull Eksp Biol Med. 1985 Jul;100(7):57-60.

PMID:3926018
Abstract

The soft tissues around the implant with a suspension of P. aeruginosa were studied by light and electron microscopy. Severe damages to structures of the histohematic barrier, phagocytes were revealed as was the bacterial invasion into thrombosed vessels of the microcirculatory bed. The mechanism of the micrometabolism as playing the key role in the bacterial penetration from the primary septic focus into the common bloodstream is discussed.

摘要

通过光学显微镜和电子显微镜对植入物周围带有铜绿假单胞菌悬液的软组织进行了研究。发现组织血液屏障结构受到严重破坏,吞噬细胞出现,细菌侵入微循环床的血栓形成血管。讨论了微观代谢在细菌从原发性脓毒症病灶进入全身血液循环中起关键作用的机制。

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