Hussain Syed H, Zaidi Mohammed, Zaidi Mariam, Grabau Guy M
College of Osteopathic Medicine, Kansas City University, Joplin, USA.
Department of Internal Medicine, Freeman Hospital West, Joplin, USA.
Cureus. 2024 Sep 12;16(9):e69273. doi: 10.7759/cureus.69273. eCollection 2024 Sep.
Hypomagnesemia can occasionally present with severe neurological deficiencies, and it is usually attributed to an underlying renal and/or gastrointestinal pathology. Rarely, patients may present with neurological symptoms in the absence of an obvious cause. Our case highlights the importance of considering hypomagnesemia as a primary cause of those presenting with severe neurological deficits in an intensive care unit setting, as well as the significance of conducting a thorough social and medical history on these patients to elucidate their underlying causes. We discuss the case of a 48-year-old Caucasian male who acutely presented with seizures, tremors, visual hallucinations, diplopia, personality changes, and ataxia with recurring severely low magnesium (0.4 mg/dL) at times in the absence of renal, gastrointestinal, hormonal, infectious, or autoimmune pathology.
低镁血症偶尔会表现为严重的神经功能缺陷，通常归因于潜在的肾脏和/或胃肠道病变。很少有患者在没有明显病因的情况下出现神经症状。我们的病例强调了在重症监护病房环境中，将低镁血症视为出现严重神经功能缺陷患者的主要病因的重要性，以及对这些患者进行全面的社会和病史调查以阐明其潜在病因的意义。我们讨论了一名48岁白种男性的病例，该患者急性发作时出现癫痫、震颤、视幻觉、复视、人格改变和共济失调，且在没有肾脏、胃肠道、激素、感染或自身免疫病变的情况下，有时会反复出现严重低镁（0.4mg/dL）。
